Aortic coarctation is one of the most critical cardiovascular diseases. Acute and severe pain is the main clinical manifestation. There are two major medical classifications of aortic coarctation. The most widely used is the 3-type classification proposed by Prof. DeBakey et al, in 1965. Type I: Aortic coarctation involves the ascending aorta to the descending aorta and even to the abdominal aorta. Type II: Aortic coarctation involving only the ascending aorta. Type III: Aortic coarctation involving the descending aorta, such as downward involvement of the abdominal aorta is type IIIA; downward involvement of the abdominal aorta is type IIIB. In 1970, Professor Daily of Stanford University and others proposed another classification method based mainly on the location of the proximal endothelial fissure: Stanford type A: equivalent to DeBakey type I and II, Stanford type B: equivalent to DeBakey type III. type B: equivalent to DeBakey type III. In practice it can manifest itself in different situations, also called clinical symptoms, which mainly include some of the following: 1. Typical patients with acute aortic coarctation often present with sudden, severe, chest and back, tearing-like pain. In severe cases, heart failure, syncope, or even sudden death can occur; most patients are accompanied by uncontrollable hypertension; 2. Occlusion of aortic branch arteries can lead to corresponding ischemic symptoms in the brain, limbs, kidneys, and abdominal organs: such as cerebral infarction, oliguria, abdominal pain, pale legs, weakness, florid spots, and even paraplegia. 3. In addition to the above main symptoms and signs, because of the wide area of aortic blood supply, the performance varies according to the accumulation range of the entrapment. Other conditions include: disappearance of peripheral arterial pulsation, vocal cord paralysis when the left recurrent laryngeal nerve is compressed, hemoptysis and vomiting of blood when the entrapment penetrates the trachea and esophagus, superior vena cava syndrome when the entrapment compresses the superior vena cava, dyspnea when the trachea is compressed, and Compression of the cervicothoracic ganglion results in Horner syndrome, compression of the pulmonary artery results in pulmonary embolism, and involvement of the mesenteric and renal arteries by the entrapment may cause intestinal paralysis or even necrosis and renal infarction. Pleural effusion is also a common sign of aortic coarctation, mostly on the left side. The greatest danger of aortic coarctation is death. The aorta is the main blood vessel of the body, subjected to pressure directly from the beating heart, with tremendous blood flow, and the presence of a tear in the intimal layer, without proper and timely treatment, has a very high chance of rupture and a very high mortality rate. Previous literature reported mortality rates of up to 50% within 1 week and between 60-70% within a month. In addition to this, even if the patient survives, the enlargement of the false lumen and the increase in pressure, which reduces the blood flow to the true lumen vessels, leads to ischemia of the organs in the area supplied by the aorta. The main treatment options for aortic coarctation include conservative, interventional and surgical treatment. Interventional endoluminal repair techniques have enriched the treatment of aortic coarctation and have made the procedure less invasive and safer.