Gastric weakness is a disorder of gastric emptying in the absence of obstruction. The pathogenesis of this disorder is not well understood and may be related to a variety of factors. Psychological factors such as stress and fear after gastric surgery cause stress reactions and phytodysfunction in patients. The release of catecholamines from sympathetic nerves, which inhibit smooth muscle cell contraction, can delay gastric emptying. The trauma of the operation itself, the prolonged operation time, and the alteration of the gastric environment due to the flow of bile and pancreatic juice through the residual stomach all cause damage to the stomach and lead to gastric weakness. Some studies suggest that disturbances in the electro-mechanical activity of the stomach are also responsible for gastric weakness, as well as vagus nerve damage and dissection, which affects the recovery of gastric tone after surgery. In addition, inflammation and edema of the stomach interfere with the normal emptying of the remnant stomach, as well as altered secretion function of gastrointestinal peptide hormones, resulting in reduced remnant gastric motility. Recently, improper diet after gastric surgery is also a major trigger for the development of gastric weakness. Gastric weakness occurs mainly after Bi-II anastomosis, with an incidence of 1 to 3%, and Bi-I anastomosis can also occur. Once gastric weakness occurs, it has a long course, slow recovery, and is difficult to handle, so it should be avoided as much as possible. Preoperatively, do a good job in psychological work to relieve the patient’s fear and nervousness about surgery and ensure that the patient has sufficient rest before surgery. Pay attention to the surgical technique during the operation, be gentle and avoid unnecessary stimulation and injury. Try to adopt simple surgery according to the specific situation. Improve the general condition of the patient before surgery, correct anemia and hypoproteinemia, and encourage the patient to leave bed early after surgery. In patients with vagus nerve injury, the duration of gastrointestinal decompression should be extended appropriately. Pay attention to postoperative nutritional support and acid-base balance of water and electrolytes. Take good care of the patient’s pharynx and oral cavity. Studies have concluded that gastric incompetence is a functional disease with non-mechanical obstruction, and the traditional treatment is mainly based on a non-surgical comprehensive approach. Strict water and food fasting, continuous gastrointestinal decompression, abdominal hot compresses, and warm hypertonic saline gastric lavage can reduce anastomotic edema. Water, electrolyte and acid-base balance are maintained, and TPN support is given if necessary for slow recovery. Gastrointestinal motility drugs can promote the contraction of the ventricle of the esophagus; erythromycin administered intravenously can promote the contraction of the gastric wall; morpholine can promote gastric peristalsis and emptying; cisapride and neostigmine can promote smooth muscle contraction. Jones MP et al. reported that patients with gastric weakness had to be treated non-surgically for 21-63 days for remission. Recently, some foreign articles suggest that gastric weakness, also known as gastric shock, is related to the lack of electrical stimulation in the stomach, so low-frequency electrical stimulation of the stomach with gastric weakness can partially restore the peristaltic function of the stomach and achieve certain results. On the one hand, gastroscopy can stimulate gastric peristalsis, and on the other hand, the placement of nutrition can play a guiding role, allowing gastric juice to enter the small intestine along the nutrition tube. At the same time, after the nutrition tube was placed, enteral nutrition support was given, which is in line with the patient’s physiology and stimulates intestinal peristalsis, and at the same time, enteral nutrition support can reduce the edema of the gastrointestinal tract and promote the patient’s recovery, the average recovery time of this group is 2~3 weeks, which significantly reduces the patient’s hospital stay compared with the traditional passive treatment method. The placement of gastroscopy-guided nutrition tube is easy and safe to operate, and the effect is clear, which is a more effective method to treat gastric weakness.