Sleep disorders are manifestations of abnormal sleep volume and abnormal behavior during sleep, as well as disturbances in the normal rhythmic alternation of sleep and wakefulness. Can be caused by a variety of factors often associated with physical illness. Insomnia is just a symptom, in addition to neurasthenia, depression, anxiety people may have this symptom. If you subjectively put yourself into the population of neurasthenia, without carefully looking for the cause, it is likely to delay the restoration of good sleep treatment. World Sleep Day, a 10,000 people questionnaire survey showed that 45% of the people in the country have different degrees of sleep disorders. Sleep disorders have two manifestations, one is the difficulty of falling asleep, if lying in bed for 30 minutes and still can not sleep, and maintained for a period of time, may be a problem. In addition, sleep disorders are also reflected in the maintenance of sleep, some people often wake up at three or four o’clock in the morning, and can no longer sleep, or sleep a night to wake up several times, which also belongs to a kind of sleep disorder. There are a number of external causes of sleep disorders, and environmental factors are an important one. A room that is too bright, too noisy, or too dry in the winter when the heat is on can all affect the quality of sleep. In addition, more patients hinder a good night’s sleep because they are stressed at work, too tired and always thinking about things that don’t go their way in life. Overcoming sleep disorders should start from these aspects. First, give yourself a comfortable space to sleep, the bed should be comfortable, and it is best to hang curtains with good light-blocking effect in the bedroom, and at the same time, seal the doors and windows well, so as to save the noise from outside to disturb your rest. Secondly, winter climate is dry, put a humidifier in the bedroom will play a good role in sleep. Put a cup of water by the bedside, in case you are thirsty at night, you don’t have to get up to find water to drink, lest you feel sleepy. Thirdly, don’t take drugs that excite the central nervous system before going to bed. Coffee, strong tea and chocolate are all foods that you shouldn’t choose before going to bed. Some people think, drink some wine can help you sleep, in fact, not, many people drunk after waking up feeling weak, head also drowsy, it is the alcohol so that the quality of sleep decreased. Really belong to the neurasthenia and insomnia patients are not many. On the contrary, many people due to depression or anxiety to put themselves into the neurasthenia. These patients as long as you find the real cause of insomnia, the right medicine, take some antidepressants, anti-anxiety drugs, and then with psychotherapy is completely able to free themselves from insomnia. To sleep well, we should develop good sleep hygiene habits, and sleep regularly on time. People with sleep disorders should not take drugs indiscriminately on their own, but should take drugs under the guidance of a doctor in order to overcome sleep disorders. In addition to the above methods, Chinese medicine treatment of sleep disorders, insomnia has its own unique insights, and the efficacy is remarkable. The traditional method of treating insomnia and depression with western medicines often has high side effects and is easily addicted. Chinese medicine has the advantage that sleeping pills do not have, i.e., it is not addictive and does not cause dependence. Modernization of traditional Chinese medicine also makes traditional Chinese medicine in the field of treatment of sleep disorders and insomnia show its strength, through the selection of natural precious herbs, combination of excellent formulas for the treatment of insomnia, there are a large number of high-tech achievements of traditional Chinese medicine in the field of insomnia treatment plays an important role, such as sour jujube seed capsule, lily sour jujube seed capsule, nine flavors of the Divine An capsule, etc., coupled with the use of psychological behavioral therapy methods, the treatment of sleep disorders and insomnia certificate has achieved a Very satisfactory therapeutic effect, solve insomnia, improve sleep quality, relieve headache, dizziness, fatigue and other phenomena; relieve tension, anxiety, depression, memory loss, neurasthenia and other uncomfortable symptoms, achieved very good results, and without any toxic side effects and dependence, to help people really realize the natural health of sleep. Osteoporosis is a group of bone disease caused by a variety of reasons Bone tissue has normal calcification, calcium salts and matrix is a normal proportion of metabolic bone lesions characterized by a decrease in the amount of bone tissue per unit volume In most osteoporosis, the reduction of bone tissue is mainly due to increased bone resorption due to the onset of slow individual faster to bone pain, easy to fracture characterized by biochemical examination of basically normal pathology can be seen in the anatomy of the bone cortex is thin, trabeculae are sparse and atrophic class Bone cortex is thin, trabecular bone is sparse and atrophic, and the bone matrix is not thick. Divided into idiopathic and secondary (primary): 1, endocrine Cortisolism Hyperthyroidism Primary hyperparathyroidism Acromegaly Hypogonadism Diabetes mellitus, etc.; 2, pregnancy and breastfeeding; 3, nutritive Protein deficiency Vitamin C, D deficiency Low-calcium diet Alcoholism, etc.; 4, hereditary Osteogenesis Imperfecta Chromosome anomalies; 5, liver disease; 6, renal diseases Chronic Nephritis Corticosteroids, anti-epileptic drugs, anti-tumor drugs (e.g. methotrexate), heparin, etc.; 8. Generalized osteoporosis is seen in long-term bedridden paraplegia, space flight, etc.; localized osteoporosis is seen in post-fracture Sudecks’ muscular atrophy, post-injury muscular atrophy, etc.; 9. Gastrointestinal malabsorption, gastrectomy, etc.; 10. Rheumatoid arthritis, etc.; 11. Tumors, multiple myeloma, metastatic carcinoma, mononucleated cellular leukemia, Mast-Cell leukemia, etc. Leukemia Mast-Cell disease, etc.; 12. Other causes Smoking Bone loss Transient or migratory osteoporosis. I. Senile and postmenopausal osteoporosis After 55 years of age in men, women are seen after menopause. Senile osteoporosis may be related to low level of sex hormone, weakening of anabolic stimulation of proteins and diminishing function of osteoclasts and reduction of bone formation. Estrogen inhibits osteoclast activity, reduces bone resorption and promotes osteoblast activity and bone formation, and antagonizes the effects of cortisol and thyroid hormone. After menopause, estrogen decreases, so bone resorption accelerates and osteoporosis occurs gradually. Estrogen also stimulates l-a-hydroxylase to produce 1.25-(OH)2-D3. The lack of sex hormones after menopause, 1-a hydroxylase to parathyroid hormone (PTH) low blood phosphorus and other stimulation of the sensitivity of the generation of 1.25-(OH)2-D3 biosynthesis is low. Also involved in the occurrence of osteoporosis, with the age of osteoblasts gradually die, bone matrix in quantity and quality are changing, so senile osteoporosis is actually the performance of the aging process of the organism, especially bone tissue is the most prominent performance. B. Nutritional osteoporosis: Protein deficiency, poor generation of bone organic matrix, vitamin C deficiency affects matrix formation and impairs the maturation of collagenous tissues; dietary calcium deficiency (less than 400mg per day) can be secondary to hyperparathyroidism, and promote bone resorption can also be a cause of the disease. Wasting osteoporosis for various reasons, such as wasting, not moving, not bearing weight, etc. Mechanical stimulation of bones can be weakened, resulting in muscle atrophy, decreased bone formation, increased bone resorption, and formation of osteoporosis. 4. Idiopathic osteoporosis of young people with unknown causes, most common in young people, so it is also known as osteoporosis of young people. 5. Endocrine osteoporosis. 1. Corticotrophic hyperplasia. Inhibit intestinal calcium absorption, increase urinary calcium excretion, inhibit protein synthesis, increase decomposition, lead to negative calcium and nitrogen equilibrium, so that osteogenesis is impaired, but mainly bone resorption increases. 2. Hyperthyroidism, a large number of thyroid hormones have a direct effect on the bones, so that bone resorption and bone formation at the same time, but bone resorption is more prominent, resulting in a decrease in bone mass. Hyperthyroidism is characterized by systemic metabolism, and the lack of protein matrix in the bones. Impaired deposition of calcium salts is also a cause of low bone mineral density 1.25-(OH)2-D3 is the active hormone of vitamin D. It increases intestinal absorption of calcium and phosphorus and stimulates bone growth and mineralization Because a large amount of thyroid hormone affects the activity of renal 1-a-hydroxylase, which interferes with the catabolism of 1.25-(OH)2-D3, 1.25-(OH)2-D3 levels decrease in the presence of hyperthyroidism, resulting in reduced intestinal absorption of calcium and a decrease in fecal calcium. Decrease in intestinal calcium absorption and increase in fecal calcium excretion Decrease in renal calcium absorption and increase in renal calcium excretion Enhancement of collagen decomposition Increase in urinary hydroxyproline excretion Negative calcium balance Therefore, the decrease in bone mineral density of patients with hyperthyroidism and the decrease in 1.25-(OH)-2-D3 may be related to each other to a certain extent. 3. Diabetes mellitus Relative or absolute insufficiency of insulin leads to impaired protein synthesis Negative nitrogen balance in the body Poor bone matrix generation Poor bone amino acid reduction, impaired collagen synthesis, decreased intestinal calcium absorption, decreased bone calcification, increased urinary calcium and phosphorus excretion and impaired renal tubular calcium and phosphorus reabsorption in diabetic patients due to diabetic high glucose osmotic diuresis, resulting in negative calcium balance, causing secondary hyperparathyroidism, and increased secretion of PITll, bone decalcification, and hepatic dystrophy and renal lesions when diabetes mellitus is poorly controlled, causing decreased activity of vitamin D, decreased a-hydroxylase activity, and reduced activity of vitamin D, and increased bone calcium production. The decrease in a hydroxylase activity increases bone decalcification. 4. Acromegaly is often accompanied by adrenal gland enlargement, cortical hypertrophy, relative hyperthyroidism, and hypogonadotropic hypogonadism, resulting in the suppression of growth hormone, cortisol, and thyroid hormone, which can increase urinary calcium excretion, decrease blood calcium, and increase blood phosphorus, thus stimulating the secretion of PTH and increasing bone resorption. 5. Primary hyperparathyroidism is associated with osteoporosis, which is characterized by a decrease in PTH production and a reduction of bone mineral content in the bone, and an increase of bone resorption. Mesenchymal cells, primitive osteoblasts, preosteoblasts, osteoclasts, preosteoblasts, osteoblasts, and osteocytes are all affected. Acute experiments have proved that PTH firstly activates a large number of osteocytes to play its role of bone resorption, and at the same time promotes a few inactive preosteoblasts into active osteoclasts to accelerate the role of osteolysis and resorption. At this time, the transition from osteoclasts to preosteoblasts and osteoblasts is inhibited due to a decrease in the level of inorganic phosphorus in the cytoplasm, and osteoblasts are small and scarce, resulting in the efflux of calcium salts from the bone. However, subperiosteal resorption of the bone cortex is its characteristic change. 6. hypogonadism as mentioned above. 7. hereditary tuberculosis (1) osteogenesis imperfecta (osteogenesis imperfecta) is a kind of autosomal dominant inherited disease due to the production of bone matrix by osteoblasts is less, as in osteoporosis; (2) hemidemineralization (Homocystinaris) is mainly due to the lack of leucine synthetase, which is the main cause of the disease. (2) Homocystinaris, which is mainly due to a deficiency of the enzyme geranylgeranyl synthetase. Rheumatoid arthritis with osteoporosis is accompanied by atrophy of connective tissues, including collagenous tissues of bones. Corticosteroid therapy also promotes osteoporosis. Long-term heparin therapy affects the collagen structure, which can lead to osteoporosis. Osteoporosis prevention: 1) dietary calcium intake; 2) exercise; 3) appropriate sun exposure; 4) appropriate amount of VitD supplementation; 5) the use of estrogens; 6) calcium inhibiting hormones. Hormones. Understanding its pathogenesis, it is not difficult to find sleep disorders and osteoporosis can be avoided and reduced through changes in lifestyle and the development of good habits, on the occasion of New Year’s Eve in 2008, I would like to dedicate this article to the most I am concerned about the nightly have to go to sleep after two or three o’clock your people; as well as you sleep poorly, suffering from or have the potential for osteoporosis of friends!