Ascites can form again after being withdrawn, depending mainly on the different mechanisms of floaters formation. Commonly, ascites is mostly the result of a combination of hepatic decompensation and portal hypertension, and is a typical clinical manifestation of cirrhosis in its decompensated phase, with very large amounts of ascites and an abdomen that bulges like a frog’s belly. The mechanism of formation is that portal hypertension increases the blood pressure in the vascular bed of the intra-abdominal organs, which reduces the recovery of tissue fluid leaking into the abdominal cavity, the deficiency of effective circulating blood volume, the hypoproteinemia caused by the decompensated phase of liver function, and the decrease of colloid osmotic pressure causing the leakage of fluid from the capillaries into the abdominal cavity are also typical causes. There is also a small percentage of sodium and water retention due to inactivation of aldosterone and antidiuretic hormone by the liver, resulting in secondary aldosteronism and antidiuretic hormone increase. The ascites formed in this condition is mainly to resolve the primary condition, the decompensated phase of cirrhosis, and the treatment is only to maintain and improve the quality of survival of the patient, ascites is difficult to be completely eliminated and can be relieved by diuresis and high dose protein infusion to alleviate the generation of ascites.