The clinical symptoms of vascular Parkinson’s syndrome are mainly lower limb dyskinesia. Typical symptoms are “magnetic foot reaction” (extreme difficulty in starting), more obvious gait disturbance, and rare tremor. It is usually caused by a lacunar infarct in the basal ganglia region, and in the absence of a history of acute stroke, the clinical picture is similar to that of geriatric gait disorder. It is often accompanied by cone-bundle signs and dementia. In patients with this disease, extensive frontal white matter damage can lead to downward conduction pathway obstruction, which leads to bilateral lower limb movement disorder, senile gait disorder and cone-bundle sign; bilateral frontal white matter interconnecting fibers are damaged, which may lead to the emergence of dementia. Prevention of this disease focuses on controlling risk factors for cardiovascular disease, and early treatment of cardiovascular disease, with the administration of antiplatelet adhesion or aggregation drugs under the supervision of a physician, may be beneficial in reducing the incidence of vascular Parkinson’s syndrome. Later, drugs such as amantadine may be added to treat Parkinson’s. When symptoms related to vascular Parkinson’s occur, it is recommended to seek timely medical treatment and standardize treatment under the guidance of a physician to avoid delays.