Talk about how calcium deficiency is linked to cerebral palsy

  ”Come on, come on”, a cute little girl finally walked alone with her uncoordinated steps under her mother’s encouragement, one meter, two meters, three meters…, the more the little girl walked, the more stable she became. This is a scene that happened in Belle Building. When Tingting was one year old, compared to other children her age, Tingting was not only unable to walk alone, she could not even sit still, and she was still pointing her toes when she stood up. The doctor told her that this was a calcium deficiency and that she would be fine as long as she took calcium supplements, so Tingting’s mother bought a bunch of calcium supplements and went back, but after a month, two months, and six months, Tingting was sitting a little more steadily, and nothing had changed much. Tingting’s mother couldn’t sit still anymore, so she asked around and found our hospital. After the outpatient experts diagnosed that this was not a “calcium deficiency” at all, but a spastic cerebral palsy, which needed a comprehensive rehabilitation treatment based on points and acupuncture points in order to walk. After three courses of rehabilitation, Tingting, who could not stand or walk alone, was able to walk alone, which led to the scene at the beginning. So what exactly is “calcium deficiency” and what is cerebral palsy?
   Its clinical manifestations are mainly.
  I. Symptoms and signs
  1. Non-specific neuropsychiatric symptoms
  (1) sweating: in addition to crying, sweating during nursing activities, wet pillow, wet underwear, sweat smell odor, and clothing, room temperature, temperature is not related.
  (2) Night terrors: The slightest external movement causes waking, jumping and crying, more than three times a night. (2) Occipital baldness: Hair loss in the area due to sweating on the head and shaking the scalp back and forth.
  2.Skeletal changes
  (1) Head: Cranial softening: < 6 months, thin skull, soft front chimney edge, severe ping-pong-like sensation;
  (2) Square skull: 7-8 months Bilateral bony hyperplasia of frontal and parietal bones with symmetrical elevation, i.e. square head, saddle-shaped or cross-shaped skull in severe cases, delayed closure of fontanelle: oversized or delayed closure of fontanelle. delayed teething.
  (3) Thorax: mostly seen in children around 1 year old, rachiticrosary: bone like tissue accumulation and expansion, round bulge can be found at the junction of ribs and rib cartilage, the most obvious is the 7th to 10th ribs, from top to bottom like a bead-like protrusion, Harrisongroove, the ribs at the attachment of the diaphragm are pulled and sunken, forming a transverse groove The rib epiphysis is sunken and the sternum protrudes forward to form a chicken chest-like deformity, and the sternum is sunken into the saber, which becomes a funnel chest. “Due to the softening of the bone and muscle joint relaxation, both lower extremities of children may have femur, cavity bone and umbilical bone bending after starting to stand and walk due to weight bearing, forming severe knee inversion (“O” type) or knee valgus (“X” type ) deformity. After the child can sit and stand, the spine may be deformed due to ligament relaxation.
  3. The whole body muscles are relaxed, the muscle tone is reduced, the muscle strength is poor, the development of movement is delayed, the muscle tone of the intestinal wall is reduced, the abdomen is bulging, and it is like a frog’s belly when lying down.
  Cerebral palsy, known as cerebral palsy, is a syndrome of non-progressive brain damage and developmental defects starting from conception to infancy. The main manifestations are central motor disorders and postural abnormalities, often accompanied by varying degrees of mental retardation, epilepsy and visual, auditory, language and behavioral disorders.
  The most common causes include: prematurity, low birth weight, giant babies, neonatal asphyxia, intracranial hemorrhage, persistent neonatal jaundice, umbilical cord encirclement, placental abnormalities, amniotic fluid abnormalities, twin fetuses, multiple fetuses, hypoglycemia, intrauterine infection, intrauterine hypoxia, maternal gestational hypertension, diabetes mellitus, etc.
  Its early manifestations include.
  1, fixed posture: often due to abnormal muscle tone as a result of brain injury, such as cornscrew, frog position, inverted U-shaped posture, etc. It can be seen in the first month after birth.
  2, can not smile: such as 2 months can not smile, 4 months can not laugh loudly.
  3.Hand clenching: If 4 months old, they cannot open their hands, or their thumbs are inwardly closed.
  4.Body twisting: 3-4 months old infants with body twisting often suggest an extraconal injury.
  5.Head instability: If at 4 months of age, the head cannot be raised in prone position or cannot be held upright in sitting position.
  6.Strabismus: 3-4 months old infants have strabismus and poor eye movement.
  7.Inability to actively grasp objects: for example, 4-5 months old cannot actively grasp objects.
  8.Gaze at hands: still present after 6 months.
  9.Inability to turn over: still unable to turn over after 6 months.
  10.Does not use lower limbs: 6-7 months old does not use lower limbs to support weight briefly.
  11.Do not use one hand: 7-10 months old infants do not use one hand to grasp and play.
  12.Hands are clumsy: 7-10 months old hands have no fine motor, such as pinching small things inflexible and uncoordinated.
  13, can not sit alone: 7 months can not sit alone.
  14.Can’t grasp the station: 10 months can’t grasp the station.
  15.Can’t say goodbye to people: After 10 months, they can’t say goodbye to people.
  16.Standing on toes: 10 months old still standing on toes.
  17.Can’t take a step: After 13-15 months, they can’t take a step.
  18.Slobbering and “hand eating”: After 12 months of age, the child still drools and “eats hands”.
  19. Uncoordinated and asymmetrical movements of the limbs at any age, with one limb being shorter than the other.
  The main difference between cerebral palsy and “calcium deficiency” is that cerebral palsy has high risk factors, abnormal muscle tone and abnormal posture, and there are generally changes in MRI and CT of the head, and there is obvious motor backwardness, and some of them are also accompanied by intellectual backwardness. In contrast, a simple “calcium deficiency” has none of these conditions. If cerebral palsy is treated as “calcium deficiency”, it will not only be ineffective, but also delay the treatment and leave lifelong disability. If parents cannot distinguish between “calcium deficiency” and early symptoms of cerebral palsy, the best way is to go to a professional pediatric rehabilitation department and let the doctor make a judgment.