The liver function of infants is not yet well developed and the biliary tract is weak in eliminating bilirubin, so that bilirubin metabolism is restricted and cannot be excreted in time, resulting in the accumulation of bilirubin in the body and thus causing high jaundice in infants. As the fetus is in an oxygen-deficient environment in the uterus, a large number of red blood cells are produced in the blood to ensure the fetus’ oxygen supply, and after the birth of the fetus, the bilirubin cannot be excreted in time because the metabolic function is not yet perfect, resulting in jaundice in the newborn. Physiological jaundice in infants after birth, about 5-15 days of normal liver function work, jaundice will subside on its own, no special treatment is required. Pathological jaundice, on the other hand, should never be ignored, such as due to viral or bacterial infections that cause impaired liver cell function, mostly occurring as a result of intrauterine viral infections. Obstructive jaundice due to congenital biliary anomalies is more common with congenital biliary atresia; hemorrhagic jaundice is more severe due to blood type incompatibility between the mother and the fetus. This type of jaundice is characterized by yellowing of the skin, mucous membranes and sclera, and the color will gradually deepen and easily recur, and the child’s appetite will be seriously reduced. Neonatal jaundice should not be ignored, and there is no need to make a big deal about it. You can feed the baby plain water in moderation and follow medical advice to treat the jaundice so that the baby can get rid of it as soon as possible.