Currently, polycystic ovary syndrome is a complex endocrine disorder that clinically affects female fertility. The causes of polycystic ovaries are inconclusive, but they are related to the following factors Polycystic ovary syndrome is characterized by chronic anovulation and hyperandrogenemia, with common clinical manifestations such as abnormal menstruation (scanty menstruation, amenorrhea), infertility, hirsutism or acne. The possible causes are currently considered by medical science to be: 1. Genetic factors. Familial ovulatory dysfunction and ovarian polycystic-like changes suggest a genetic basis for the disease. Cytogenetic studies have shown that polycystic ovary syndrome may be inherited in an X-linked recessive, autosomal dominant or polygenic manner. 2. Hormonal endocrine factors. Patients with polycystic ovary syndrome are often associated with insulin resistance, hyperandrogenemia and hyperprolactinemia. These endocrine hormone disorders are an important cause of polycystic. 3. Ovarian factors. As patients with polycystic ovary syndrome have high LH, that is, luteinizing hormone, the ratio with FSH is greater than 1.5 times or more. Some scholars believe that certain substances may exist in the ovaries, which may reduce the uptake of follicle-stimulating hormone by ovarian cells, thus inhibiting the development of follicles and the formation of non-dominant follicles. 4. Obesity. The more serious obesity is, the more disturbed the internal environment is, and the greater the possibility of polycystic ovaries. Therefore, patients diagnosed with polycystic ovaries should also be actively treated. Women with fertility requirements should undergo ovulation promotion treatment under the guidance of a physician.