The tachycardia folding loop of AV node folding is located at the level of the AV node. In a normal person, AV node conduction is only one pathway and one pathway does not form a fold. For reasons that are not entirely clear at this time, there is another pathway in the AV node region that is slower than the previous pathway, called the “slow path”, and accordingly, the previous pathway is called the “fast path”. A foldback loop between the fast and slow paths may result in tachycardia. The typical atrioventricular nodal folding tachycardia impulse travels in the folding loop in the direction of antegrade conduction along the slow path (atrium to ventricle) and then inverse conduction along the fast path (ventricle to atrium), in a circular fashion, usually agitating the atrium and ventricle once per week. In a few cases, the loop may also be transmitted anteriorly along the fast path and then retrograde along the slow path, and in some patients there may even be multiple slow paths, with the loop forming between the 2 slow paths. Ablation may be considered in patients with a history of multiple episodes of tachycardia with atrioventricular node regression, significant symptoms, and in patients who have failed drug therapy or who are unwilling to undergo long-term drug therapy. Because tachycardia episodes do not occur daily or even hourly like premature beats, they are low-frequency events that may occur only once a month, several months, or even once a year, and it is controversial whether long-term medication is the best treatment for such low-frequency events to reduce the episodes. Ablation therapy for AV nodal regression tachycardia is well established and is preferred for multiple episodes of symptomatic AV nodal regression tachycardia, given the inconvenience of long-term drug use and the potential side effects of the drugs themselves. The ideal scenario for ablation of AV nodal regression tachycardia is when a slow atrioventricular node conduction is detected by cardiac catheter electrophysiology prior to ablation and AV nodal regression tachycardia is artificially induced. In this way, if the slow pathway disappears after the ablation and the tachycardia is no longer induced by repeated electrophysiological examinations, the treatment effect is more certain. If tachycardia is not induced, appropriate intraoperative medication can be given to stimulate the conduction system of the heart to increase the likelihood of tachycardia induction. Ablation may also be considered for tachycardia with a clear clinical history of supraventricular tachycardia and electrophysiological examination confirming the presence of slow conduction and slow associated folding loops, but failure to induce AV node folding. The need for ablation of AV nodal folding loops is unclear and controversial in cases where electrophysiological examination of other arrhythmias reveals the presence of AV nodal slow conduction and induces AV nodal folding tachycardia, i.e., where there is no clinical history of an episode but an AV nodal folding tachycardia is induced on electrophysiological examination. Historically, ablation of tachycardia with AV node regression has been performed by disrupting the regurgitant ring, but there are two strategies: ablation of the fast track and ablation of the slow track. The ablation of the fast pathway and the retention of the slow pathway as the conduction pathway of the AV node will result in a long conduction time of the electrical impulse in the AV node, which is not conducive to the coordinated contraction of the atria and ventricles, and the possibility of complete block of the entire AV node conduction is relatively high. The slow pathway is located posterior to the tricuspid annulus and is close to the coronary sinus opening. To ablate the slow path, an ablation catheter is placed under X-ray fluoroscopy in the approximate location of the slow path, and then the location of the slow path is ablated by fine calibration of the recorded potential. The success rate of ablation of slow trails for atrioventricular nodal regression tachycardia is over 95%. Permanent AV node block is the most common and serious complication, but the ablation strategy of the slow pathway results in a complication rate of less than 0.5%. Other complications are mainly common to some cardiac interventions.