The main treatment for carbon monoxide poisoning is to actively correct hypoxia and prevent cerebral edema, as follows: 1. Correction of hypoxia: The poisoned person should be immediately removed from the scene of poisoning, moved to fresh air and keep the respiratory tract unobstructed. Oxygen inhalation can improve the partial pressure of oxygen in patients to correct hypoxia. In addition, hyperbaric oxygen therapy should also be actively given to patients who are comatose or have had a history of coma in carbon monoxide poisoning, as well as to patients who have obvious symptoms of the cardiovascular system and have significantly increased carboxyhemoglobin. Hyperbaric oxygen therapy can increase the volume of physical dissolved oxygen in the blood for use by tissues and cells, which in turn accelerates dissociation of carboxyhemoglobin and promotes carbon monoxide clearance. Hyperbaric oxygen therapy can not only shorten the course of the disease and reduce the death rate of carbon monoxide poisoning, but also reduce or prevent the occurrence of delayed encephalopathy; 2. Prevent cerebral edema: 2-4 hours after acute carbon monoxide poisoning, patients may develop cerebral edema, and 24-48 hours to reach the peak of cerebral edema, and it can last for many days. It is recommended that drugs such as hypertonic dehydrating agents, diuretics or glucocorticoids can be applied early to prevent or treat cerebral edema so as to promote cerebral blood circulation, maintain respiratory and circulatory functions, and actively give symptomatic support treatment. In addition, patients should not be careless if they are resuscitated after resuscitation, they should be absolutely bed-rested, closely observed for 2 weeks, and care should be strengthened to detect and treat delayed encephalopathy in a timely manner.