Differential diagnosis of orbital narrowing

Meningioma of the lateral 1/3 of the pterygoid crest. The lateral type of meningioma of the pterygoid crest presents late, with the tumor occurring on the large wing of the pterygoid bone, causing bone growth in the posterior lateral wall of the orbit and the temporal area, shrinking the orbit, or the tumor invading directly into the orbit and causing the eye to protrude. What are the symptoms that are easily confused with it? 1. Wide orbital distance between the eyes The bony distance between the medial walls of the two orbits is excessively wide. The diagnosis is mainly based on the interorbital distance (IOD) measurement. 2, periorbital edema It refers to puffiness around the orbit and is usually seen in patients with edema. Check for periorbital or facial congestion and the presence of fever. Fever suggests possible acute paranasal sinusitis, cavernous sinus embolism, periorbital cellulitis, meningitis, or dengue. Congenital microphthalmia (CMIC) is a congenital abnormal developmental ophthalmology disorder that is characterized by a smaller than normal anterior and posterior diameter of the eye, narrow lid fissures, small orbits, and a deep sunken eye in the orbit. Patients usually have poor vision and are difficult to treat. 4. Narrow lid fissures Congenital narrow lid fissure syndrome is characterized by small lid fissures. It is inherited in an autosomal dominant fashion. It may be due to an imbalance between the number of inhibitors of maxillary protuberance development and the number of facilitators of external nasal protuberance development around the third month of embryonic life. As a result, there is also a widening of the inner canthus of both eyes and an external deviation of the lower tear point. It is more common in the Japanese. 5. Widening of the eye distance is one of the clinical manifestations of abnormal hemoglobinopathy. The disease is caused by genetic defects (autosomal dominant inheritance) that cause mutations in the genes of pearl proteins and abnormal peptide chain structure or impaired synthesis, resulting in one or more structural abnormalities of hemoglobin that partially or completely replace normal hemoglobin.