Pulmonary fibrosis is mainly caused by the secretion of collagen by the interstitium for self-repair when the lung is damaged. When the repair is excessive, fibroblasts will overproliferate and extracellular matrix will accumulate in large quantities. Pulmonary fibrosis needs to be treated symptomatically according to the degree of progression of the disease. In patients with primary disease, the progression of the disease can be slowed by removing the cause. Because the most common symptom of pulmonary fibrosis is dyspnea, it is often easy to ignore when the disease is mild. Therefore, once detected, pulmonary fibrosis needs to be seen at a regular hospital respiratory medicine department as soon as possible for a comprehensive examination and targeted medication according to the specific condition. For idiopathic pulmonary fibrosis, pirfenidone is preferred, while other types may take glucocorticoids, acetylcysteine with/without azathioprine, cyclophosphamide and other immunosuppressants, thus playing an antioxidant and anti-fibrotic role. Modulation with herbs is also possible, but clinical evidence is insufficient. There are no particularly effective drugs to cure pulmonary fibrosis at this time, but a more optimal functional status can be maintained through active and aggressive consultation, proper treatment, and close follow-up, along with usual life maintenance.