Adenoidal hypertrophy is a hyperplasia of the pharyngeal tonsils. Adenoid hypertrophy in children is often physiological, and the lymphoid tissue in the nasopharynx is present at birth and proliferates with age, reaching its maximum extent by the age of 6. Later, it gradually degenerates, and only if it affects general health or adjacent organs is it called adenoid hypertrophy. The cause of childhood is prone to acute rhinitis, acute tonsillitis and influenza, if repeated attacks, the adenoids can rapidly increase hypertrophy, resulting in increased nasal obstruction, obstructing nasal drainage, rhinitis sinusitis secretions and stimulate the adenoids to continue to proliferate, forming a vicious circle of mutual cause and effect. Mostly seen in children, often combined with chronic tonsillitis. Clinical manifestations (a) local symptoms: children with adenoid hypertrophy blocking the posterior nostril and pharyngeal pharyngeal opening, otorhinopharyngeal and other symptoms can occur. The symptoms include open mouth breathing during sleep, snoring with the back of the tongue, restless sleep at night, nasal secretion, occlusive nasal sound and slurred voice when speaking. Because of long-term open-mouth breathing, resulting in facial bone development disorders, the maxilla becomes longer, the hard palate high arch, dental alignment is not neat, upper incisors exposed, thick lips, lack of facial expression, with dementia performance, the formation of “adenoid face”. Ataxia between swallowing and breathing is dysfunctional, and choking and coughing often occur. Secretions downstream stimulate the respiratory mucosa, prone to bronchitis. Due to the obstruction of the eustachian tube, it is easy to cause non-suppurative otitis media resulting in hearing loss and tympanic membrane invagination. (B) systemic symptoms: there are often systemic nutritional and developmental disorders, mainly manifested as chronic toxic reflex neurological symptoms, such as sluggish expression, chest tightness and restlessness, poor lung expansion, resulting in chicken chest or flat chest over time. A few due to chronic nasal obstruction, long-term hypoxia and pulmonary heart disease, and even acute heart failure. The examination shows adenoid face, high and narrow hard palate, pink, lobulated lymphoid tissue block at the top of nasopharynx on postnasal rhinoscopy, soft masses can be palpated in the nasopharynx, and lateral nasopharyngeal radiographs can be taken if necessary to help diagnosis. (3) Note: Snoring caused by adenoid hypertrophy in children is often neglected by parents, but it mostly constitutes the cause of snoring together with tonsillar enlargement, so special attention should be paid to the presence of apnea, and adenoids should be examined in hospital if necessary. The adenoids in children can be examined indirectly under nasopharyngoscope, and more advanced fiberoptic laryngoscope or electronic laryngoscope and nasal endoscope can easily examine the adenoids. Treatment Surgical removal of adenoids is recommended for children over 3 years of age, and is contraindicated for children under 2 years of age (the youngest I have done is 2 years and 7 months). It is recommended that the post-operative application of herbal medicine to regulate the immunity of the child is more beneficial.