Common orbital foreign bodies include metal shrapnel, steam gun shells, or wood or bamboo fragments. There may be local swelling and pain. If combined with purulent infection, it can cause orbital cellulitis or fistula. Since most intraorbital metal foreign bodies are wrapped in soft tissues and there are fine nerves, blood vessels and muscles in the deep orbital area, it is not necessary to remove such foreign bodies from the deep orbital area. Vegetative foreign bodies can cause chronic septic inflammation and should be removed as soon as possible. Intraorbital foreign bodies enter the orbit from the front, mostly through the eyelid or conjunctiva, and enter the deep orbit between the eyeball and the orbital wall, while a few enter the orbit through the double perforation of the eyeball. Most of the orbital foreign bodies enter the orbit from the front between the eyeball and the orbital rim, and the eye may not be directly injured and most of them retain their vision. Injury-causing objects are mainly metallic foreign bodies such as lead bullets and steel, but a few are non-metallic such as glass and plants. Optic nerve disease: The optic nerve is the segment of the optic pathway from the optic disc to the optic cross, which is divided into the intra-orbital, intra-orbital, intra-osseous canal and intracranial segments. The optic nerve is surrounded by three layers of sheaths, which are continuous with the dura and arachnoid in the skull. The three sheaths and the wall of the eye fuse behind the sphere to form a blind canal, which forms two lacunae with the subdural and subarachnoid cavities in the skull. The outermost layer of the sheath is rich in sensory nerves. Therefore, when the optic nerve is inflamed, there is pain when the eye is turned or compressed. The pathogenesis of the optic nerve is closely related to its structural characteristics: the optic nerve fiber bundles are surrounded by a rich capillary network, so the optic nerve fibers can become inflamed and toxic under the action of infectious and toxic substances, and the central retinal vessels enter the eye via the optic nerve, causing blockage of axoplasmic flow in the axons of the ganglion cells in front of the scleral sieve plate due to intraorbital swelling or increased intracranial pressure, resulting in significant swelling of the axons and leading to optic papillar edema. This leads to optic papillary edema; when intraocular pressure is increased, it can cause glaucomatous optic papillary depression and atrophy. The disease is an ophthalmologic condition in which a nucleated nucleus grows within the lid and is not red or painful. This name is due to the fact that the nucleated nodules are mainly caused by phlegm and dampness blocking the veins of the lid. The disease usually has a long duration and is prone to recurrence. It is equivalent to the Western medical term chalazion. The disease is usually caused by the heat and phlegm in the spleen and stomach due to the wanton consumption of hot food, which causes the phlegm and heat to intertwine and block the meridians, resulting in the blockage of qi and blood, which is then hidden in the lid. It can also be caused by a needle eye in the eyelid that has not become pus, or by pus that has become pus but does not break down.