Traumatic asphyxia is a relatively rare syndrome in chest trauma, and its incidence accounts for about 2% to 8% of chest injuries. Common causes of injury include landslides, house collapses, crushing in car accidents and injuries in mass stampede events. When the chest and upper abdomen suffer a strong external force in an instant, the human body will instinctively reflexively hold its breath, resulting in a sudden tightening of the vocal cords, the trachea and lung air can not overflow, the result of the simultaneous action of two factors, causing a sudden increase in intrathoracic pressure, compression of the heart and large veins. Since the human superior vena cava system has no venous valves, the sudden high pressure can cause the blood from the heart to flow backwards toward the head and face, thus causing venous overfilling, blood stasis, extensive capillary damage, punctate hemorrhage, and in severe cases, even small venous rupture hemorrhage can occur. Traumatic asphyxia is mostly seen in adolescents and children with good thoracic elasticity, and most are not accompanied by chest wall fractures. However, when the external force is too strong, in addition to sternal and rib fractures, there may also be intra-thoracic or intra-abdominal organ injuries, as well as spinal and extremity injuries, and respiratory distress or shock may occur. In traumatic asphyxia, there are hemorrhagic bruises or bruises on the subcutaneous tissues of the head, neck, upper chest and upper extremities, oral mucosa and conjunctiva, and in severe cases, the skin and conjunctiva are purple and swollen. The slight intracranial punctate hemorrhage and cerebral edema can produce cerebral hypoxia, which can cause transient disorders of consciousness, dizziness, head swelling, irritability, and a few have twitching of limbs and hyperactive tendon reflexes. If intracranial hemorrhage or hematoma occurs, it may cause hemiparesis, coma or even death.