Cardiac stenting is important for the treatment of acute myocardial infarction and some angina pectoris. Through stenting, coronary arteries that have reached a certain degree of stenosis (generally 75% or more) or occlusion are reopened, and blood flow is reopened to relieve myocardial ischemia, save the dying myocardium, and protect heart function. However, this is only a physical or mechanical solution to some of the already narrowed and symptomatic coronary artery lesions, but has no effect on the generation and restenosis of atherosclerotic plaques. If prevention and treatment are not strengthened, plaques (including in-stent) will continue to grow and form new ischemic lesions, leading to serious consequences. Therefore, stenting is not a “cure” for coronary artery disease, and long-term treatment with statins and aspirin is still needed. The main function of statins (such as atorvastatin, resulvastatin, simvastatin, fluvastatin, etc.) is to reduce the level of cholesterol synthesis in the body. This is because cholesterol is an important component of atheromatous plaque. Continuing to take statins after stenting helps to keep cholesterol at a lower level and helps to reduce plaque formation. In addition, statins can stabilize plaque and prevent plaque rupture; anti-inflammatory and other effects. Therefore, it is important to take statins and aspirin under the guidance of cardiologists in regular hospitals for a long time after heart stenting. Never stop the medication without permission.