Uterine adhesions are fibrosis and deformation of the uterus caused by damage to the endometrium for various reasons, leading to decreased menstruation, infertility and miscarriage in women. Prevention of recurrence of uterine adhesions is a key issue, and recurrence of uterine adhesions is one of the independent influences on the patient’s inability to have a normal pregnancy and delivery after surgery. The traditional methods of preventing recurrence of postoperative cavity adhesions include intrauterine device, balloon, with high-dose estrogen or artificial cycle therapy. However, recurrence of cervical adhesions is still an open problem, with recurrence rates of up to 40-60% for severe cervical adhesions. What is the amniotic membrane on the human placenta? It is a translucent membrane-like substance between the fetus and the placenta that is as thick as cling film. It is a special low immunogenic substance that does not cause an immune reaction when placed in a foreign body. The amniotic membrane has special structural and secretory functions that promote epithelial differentiation and growth, reduce inflammatory responses, reduce scar formation and prevent adhesion formation. Amniotic membrane has been introduced into surgery for over 100 years. Since 1910, when it was first used in skin grafts, it has been used in a variety of surgical disciplines, such as dermatology, ophthalmology, surgery, and neuroscience. In the field of gynecology, the main applications are artificial vaginoplasty and cervicoplasty. A literature search revealed that only one foreign medical institution has introduced amniotic implantation in the treatment of uterine adhesions since 2006, with two published articles. This new technique is considered to be more advantageous and significantly different than traditional methods. In layman’s terms, the mechanism of action of amniotic membrane implantation in preventing adhesions is twofold: first, the promotion of endometrial growth; and second, the barrier effect of the amniotic membrane, which inhibits the inflammatory response and inhibits the formation of fibrotic scarring due to the close attachment of the amniotic membrane to the uterine wall.