The presence of fecal-like vomit is a clinical symptom of acute pancreatitis. Acute pancreatitis (acutepancreatitis, AP) is a relatively common type of acute abdomen, and its incidence accounts for the third to fifth highest incidence of acute abdomen. More than 80% of these patients have a mild condition, i.e. acute edematous pancreatitis, which can be cured by non-surgical procedures and is basically an internal disease. What does the prevention of the main causes of fecal-like vomit include? 1, pancreatic abscess after 2 to 3 weeks of onset, such as secondary bacterial infection, can form abscesses in and around the pancreas, with pain, fever and systemic symptoms of toxicity, local examination with mass and pressure pain. In severe cases, sepsis may occur with persistent hyperthermia. 2, pancreatic pseudocyst late onset (about 3 to 4 weeks) by pancreatic fluid and necrotic tissue in the pancreas itself or its surrounding wrapped and become, cyst size varies, small only a few millimeters, no symptoms and signs, large up to tens of centimeters, and can even occupy the entire abdominal cavity, causing the corresponding compression symptoms such as portal hypertension, jaundice, intestinal obstruction, hydronephrosis, etc., cyst penetration can cause pancreatic-derived ascites. 3, pancreatic peritonitis contains active pancreatic enzymes exudate into the peritoneal cavity, which can cause chemical peritonitis. Exudative ascites appears in the abdominal cavity. If secondary infection can cause bacterial peritonitis. 4, respiratory insufficiency due to severe abdominal distension, abdominal pain, the patient’s diaphragm movement is restricted, pancreatic exudate can enter the thoracic cavity through the diaphragmatic fissure to form pleural fluid, damage to the alveoli by phospholipase A and the hemolysis lecithin generated under its action, increased free fatty acids damage the alveolar capillary wall, increased vasodilation caused by increased bradykinin and its increased permeability, the hypercoagulable state formed by abnormal coagulation function leads to pulmonary vascular The above factors can lead to decreased pulmonary compliance, pulmonary atelectasis, pneumonia, pulmonary depression, pulmonary edema, pulmonary infarction, and patients with dyspnea, cyanosis, and eventually acute respiratory distress syndrome (ARDS). 5, arrhythmia and cardiac insufficiency due to reduced blood volume, insufficient myocardial perfusion and the effect of myocardial inhibitory factors, as well as damage to the myocardium by toxins in the case of concurrent abscess and sepsis, patients may experience myocardial ischemia and damage, manifesting as arrhythmia and heart failure. 6. Acute renal failure is caused by renal ischemia and hypoxia. It is related to hypovolemia, shock, severe infection, electrolyte disorders and abnormal blood coagulation caused by pancreatic enzymes, the emergence of hypercoagulable state and the generation of microcirculatory disorders leading to disseminated intravascular coagulation. 7. Pancreatic encephalopathy is caused by a large amount of active protein hydrolase and phospholipase A entering the brain and damaging brain tissue and blood vessels. Patients may show neuropsychiatric abnormalities, such as hallucinations, fantasies, depression, fear, delirium, irritability, speech disorders, disorientation or lack of orientation, ataxia, confusion, coma; there may be tremors, hyperreflexia or loss of reflexes, hemiparesis, etc. EEG often has abnormalities. 8. Diabetes mellitus is caused by the destruction of pancreatic beta cells and reduced insulin secretion. Diabetic ketoacidosis and hypertonic coma may occur in severe patients. 9, hypocalcemia mechanism is: a large amount of calcium deposited in the fat necrosis area, caused by fatty acid binding to form calcium soap; increased secretion of calcitonin; severe pancreatitis can appear hypoalbuminemia, total calcium measurement can be reduced; calcium – parathyroid hormone axis is out of balance, the latter’s responsiveness to low blood calcium is reduced; calcium is transferred to fat, muscle and liver tissue. Blood calcium is significantly lower, below 1.75 mmol/L (7.5 mg/dl), and does not rise for several days, when the prognosis is often poor.