Cerebral palsy (CP), or cerebral palsy for short, is a syndrome caused by non-progressive brain damage and developmental defects in the brain from conception to infancy, mainly in the form of motor deficits and postural abnormalities. However, it is not only cerebral palsy, but also spinal muscular atrophy. Congenital muscular dystrophy and other diseases. Spinal muscular atrophy (SMA) refers to a group of diseases in which muscle weakness and muscle atrophy are caused by degeneration of predominantly anterior horn cells of the spinal cord. It was first reported by Werdnig (1891) and Hoffmann (1893) and is therefore also known as Werdnig-Hoffmann disease. The disease can be divided into 4 types according to the age of onset and the degree of lesion: Types I-III are called childhood SMA, which is an autosomal recessive disorder with a population incidence of 1/6000 to 1/10000, and is the most common fatal genetic disease in infancy. The main clinical manifestations are lower motor neuron, progressive, symmetric muscle weakness and atrophy. Congenital myotonic dystrophy begins in the neonate and presents with posterior spinal protrusion, decreased muscle tone, often with hip dislocation, proximal joint contracture, and oblique neck, while the distal joints show laxity and hyperelasticity. In some cases, excessive distal joint laxity can also be absent, while such joint contractures can occur progressively, develop, and eventually affect the ankle, wrist, and fingers that previously exhibited laxity. Patients have a wide variability in their maximum motor ability, with some patients being able to move freely and others never being able to walk independently. All three disorders can manifest as motor developmental delays, so the differential diagnosis should be based on growth and family history, tendon reflexes, electromyography, or muscle biopsy.