In China, flow dissection accounts for more than 50% of surgical treatment of portal hypertension (except for liver transplantation) [4]. Traditional flow dissection emphasizes complete blockage of the source of blood flow to the varices (with particular emphasis on the left gastric vein and its paraesophageal vein that communicates with the odd vein), thus preventing hemorrhage from ruptured esophageal varices while maintaining high portal venous pressure and maintaining portal perfusion to the liver to avoid further deterioration of liver function. However, persistent postoperative hypertension in the portal venous system also relatively promotes the establishment of esophagogastric fundic collateral circulation and the reformation of varices. The rate of postoperative rebleeding is higher than that of shunts, with an incidence generally around 10% [5-7]. Changes in FPP have been an important hemodynamic observation in portal hypertension and are thought to be the direct cause of ruptured bleeding from the varices of the esophagogastric fundus [8]. Numerous hemodynamic studies of the portal venous system have shown [9,10] that the reduction of blood flow back into the portal vein via the splenic vein after splenectomy causes a significant decrease in FPP, and that FPP may rebound slightly or even be significantly higher than before splenectomy after complete blockage of the vessels surrounding the esophagogastric fundus. In order to improve this postoperative FPP hypertension, we made an intraoperative effort to preserve the pathologically dilated compensatory shunting pathway that naturally developed during the course of the disease, the paraesophageal vein and its traffic branch with the coronary vein around the esophagogastric area; in addition to the open umbilical vein, the abdominal wall and the retroperitoneal venous traffic branch. At the same time, the penetrating branch veins formed by each traffic branch – the gastric branch, esophageal branch, high esophageal branch and ectopic high esophageal branch that enter the wall of the fundus and lower esophagus – are ligated and disconnected one by one in order to achieve complete blockage of blood flow in the esophagogastric fundus region with bleeding risk The procedure was carried out with the help of FPP. The FPP measurement before and after the surgery proved that the FPP of patients with CTP grade A, B and C in the selection group decreased after surgery, and there was a significant difference compared with the preoperative period (P<0.01); while only patients with grade B in the traditional group had a decrease in FPP after surgery, and patients with grade C had an increase in FPP after surgery; and patients with different CTP grades in the selection group had a significant decrease in FPP after surgery, and there was a significant difference compared with the traditional group (P<0.01). significant difference (P<0.01). The decrease of FPP in the selection group was more obvious than that in the traditional group, which is a reflection of the body's own compensatory shunt function and the increase of fluid efficiency after preserving the pathological shunt and blocking the penetrating branch to change the turbulent flow to laminar flow in the varicose vein. There was no significant difference in liver function between the two groups of patients one week after surgery, except for some individual indexes, compared with the preoperative period. There were no recent postoperative complications such as hepatic encephalopathy, which is mostly seen after bypass, in both groups, and the complication rate was significantly lower in the selection group than in the conventional group (P<0.01), and the mortality rate was lower (P<0.01). All of these clinical observations demonstrated the superiority of natural compensatory shunts in the paraesophageal veins. In conclusion, preserving the naturally formed, pathologically dilated, compensatory shunt of the paraoesophageal vein and using the naturally formed shunt pathway during the course of the disease to relieve the hypertensive state of the portal venous system can more effectively reduce FPP, thus definitively controlling ruptured variceal bleeding and reducing the occurrence of recent complications such as postoperative rebleeding and hepatic encephalopathy. The procedure is simple, efficacious and easy to promote clinically.