Dural arteriovenous fistulae (DAVF) is a rare intracranial vascular malformation that accounts for 10% to 15% of all intracranial vascular malformations. Most scholars usually consider DAVF to be an acquired disease. However, the exact cause of its occurrence is unknown and may be related to congenital factors, venous sinus thrombosis, surgery, trauma, venous hypertension, and vascular endothelial growth factor. TDAVF is a very aggressive form of DAVF, with venous drainage through the soft meninges and venous tumor-like expansion being the growth features of TDAVF, causing a high risk of hemorrhage and progressive neurological dysfunction. 31 cases reported by Lawton The treatment of DAVF is mainly aimed at blocking the draining veins to avoid bleeding and aggravating neurological dysfunction. The current treatment of DAVF includes transarterial embolization, transvenous embolization, radiotherapy, surgery and various combinations of these methods, but for the treatment of TDAVF, most scholars believe that surgery is one of the most effective and thorough methods. The main goal of surgical treatment is to deal with the draining vein, and blocking the draining vein can achieve the purpose of cure. Therefore, preoperative whole brain angiography should be carefully analyzed to understand the hemodynamic situation of the lesion’s blood supply artery, fistula site, and draining veins, and to clarify the origin and direction of blood flow in the draining veins. Each of them has its own unique drainage pattern and surgical access. Dissection of these arterialized veins can normalize the pressure in the intracranial venous system, reduce cerebral edema, and lower intracranial pressure without causing impaired venous blood return. The fistula and the draining vein are carefully separated under the microscope along the cortical drainage vein and the surface of the skull, and the fistula is blocked or clamped at the fistula, without removing the fistula, because the fistula is located in the cerebellar curtain, and after blocking the refluxing vein, the fistula will not rupture and will quickly become occluded by thrombosis.