For many years, many perinatal risk factors have been considered to be associated with the development of cerebral palsy, mainly including: prematurity and low birth weight, cerebral hypoxia-ischemia, birth injuries, congenital abnormal brain development, nuclear jaundice, and congenital infections. In recent years, the etiology of cerebral palsy has been discussed more thoroughly both domestically and externally, and it is agreed that abnormal development in the early embryonic stages is likely to be an important cause of prematurity, low birth weight and susceptibility to events such as perinatal ischemia and ischemia. Such developmental abnormalities in the early embryo are mainly derived from the maternal internal and external environmental influences before and after conception, genetic factors, and early gestational placental amniotic membrane inflammation caused by diseases during pregnancy. The current causes of cerebral palsy have been summarized in the literature and include: parental smoking, alcoholism, drug abuse, maternal psychiatric disorders, diabetes, vaginal bleeding, gestational hypertension syndrome, placenta praevia, pre-eclampsia, pre-eclampsia miscarriage or use of birth control pills, drugs for infertility, birth control pills, etc.; high birth rate, high pregnancy rate, history of stillbirth and stillbirth, history of preterm birth, miscarriage, twin or multiple births, fetal growth retardation, intrauterine infection intrauterine distress, placental abruption, placental dysfunction, heavy pregnancy reaction, cord bypass, emergency delivery, improperly assisted delivery, forceps delivery, breech delivery, prolonged labor, preterm or overdue delivery of low birth weight infants, postnatal asphyxia, aspiration pneumonia, hypoxic ischemic encephalopathy, nuclear jaundice or delayed jaundice, intracranial hemorrhage, head trauma, convulsions, infection, poisoning and malnutrition.