The abnormal heart sounds do not affect our life greatly, but if left untreated for a long time, they may complicate many symptoms and seriously affect our work and life. What is the difference between an abnormal heart sound and a normal heart sound?
Normal heart sound: The first heart sound is low and long, and the apical part of the heart is the loudest. The interval between one and two is short, and the apical beats are in phase at the same time. The second heart sound is high and short, with the base of the heart being the loudest. The interval between two and one is long, and the apical beats are in antitime-phase.
Sinus tachycardia: anemia, hyperthyroidism and fever, heart inflammation, heart failure and shock. Emotional excitement and exercise, adrenaline overload of the heart rate.
Sinus bradycardia: intracranial hypertension block jaundice, low A coronary heart myocarditis. Drug affects insulin, strong body heart rate slow. Both heart sounds increase at the same time normal people exercise or excitement, both heart sounds increase at the same time. Hypertensive disease anemia, hyperthyroidism fever is also the same.
The first heart sound is enhanced: ventricular large before the failure of fever hyperthyroidism, premature beats “with drugs” a sound strong. Mitral valve narrowing “beat-like”, atrioventricular block “cannon-like”.
Second heart sound enhancement: P2 enhancement mitral valve narrowing, emphysema and left heart failure. In left-right shunt precordial disease, pulmonary artery pressure is high and atherosclerosis is often present.
Diminished first heart sound: incomplete closure of the second main valve, heart failure, inflammatory infarction a low sound.
The second heart sound is diminished: arterial valve leakage or stenosis, arterial pressure low a sound failure.
Pendulum rhythm: pendulum fetal heart rhythm severe, myocarditis infarction cardiomyopathy
First heart sound splitting: one tone splitting apical clearing, electrical delayed right bundle block Pulmonary hypertension right heart failure, mechanical delay and formation.
Second heart sound split: usually split with features, longest seen in adolescents. Exhalation disappears suction is evident.
Sinus arrhythmia: Sinus rhythm is slightly arrhythmic, with normal heart sounds into cycles. Inhalation accelerates and exhalation is slow, non-disease in healthy children.
Premature beats: Pre-phase contractions are called premature beats, with ventricular premature beats being the most frequent. Atrial junctional is three in total and easy to differentiate on ECG.
Atrial fibrillation: Atrial fibrillation is characterized by three different, fast and slow unevenly. The intensity is unequal and irregular, and the pulse rate is definitely lower than the heart rate.
Physiological murmur: physiological murmur level is small, soft blowing wind is not conducted. Short duration without tremor, more common in children should be kept in mind.
Mitral valve closure insufficiency: mitral valve leakage has characteristics, rough blowing wind is decreasing. More than three levels of contraction accounted for, the left axillary conduction left lying clear, inspiratory weakening exhale obvious.
Mitral stenosis: mitral valve narrow murmur break, diastolic rumble low restriction. A hyperacoustic P2 strong, open valve sound with tremor.
Aortic stenosis: aortic narrowing is characteristic, coarse constriction sound like a saw. Incremental decremental neck transmission, A2 attenuated with tremor.
Aortic valve incompetence: aortic valve incompetence is characteristic, and the diastolic sigh is decreasing. The left apical transmission under the sternum, the second region is clearer anteriorly inclined sound, easily heard by holding the breath at the end of expiration.
Pulmonary valve stenosis: pulmonary valve stenosis is characteristic, rough constriction sound is congenital. The murmur is increasing and decreasing, and the P2 is attenuated with tremor.
Relative pulmonary valve insufficiency: pulmonary valve stenosis is characteristic, and the murmur is mostly relative. Soft and blowing wind lying inspiration clear, mitral valve narrow often combined.
Relative tricuspid valve insufficiency: tricuspid valve area has shrinkage sound, murmur nature like blowing wind. Most relative shutoff insufficiency, very few are organic.
Atrial septal defect: atrial septal murmur is characteristic of the second intercostal space on the left edge of the sternum. The murmur is blowing wind-like in the systolic phase, and the P2 split is mostly fibrillation-free.
Ventricular septal defect: ventricular septal murmur is characteristic, three or four intercostal spaces on the left edge of the sternum. Loud and rough systolic murmur, often with systolic tremor.
Arteriovenous ductus arteriosus is not closed: the continuous murmur is characteristic, rough and resembles a machine sound. When the arterial duct is not closed, it is heard near the left second rib of the chest.
Pericardial friction sounds: continuous murmur with characteristic, pay attention to identify the chest friction. Anterior tilt breath hold easy to hear, infarction pericarditis uremia.
In summary, it is still possible to see the difference between normal heart sounds and abnormal heart sounds. If patients find or check out the abnormal heart sounds, due to timely identification of the pathogen, and find a solution, or through drug therapy early to improve.