Traumatic epilepsy can be treated for the most part because traumatic epilepsy builds on a certain genetic background for seizures to occur. So for some patients with high risk factors, they can be prevented after the trauma, such as puncture wounds, firearm injuries, heavy closed craniocerebral injuries, or even extra heavy closed craniocerebral injuries, depressed fracture, and these patients can be given 1-2 weeks of medication after the operation for prevention, to avoid the formation of traumatic and epileptic epilepsy. But once the formation has occurred, regular medication can be given, and after regular medication, about 70% of patients can get a seizure-free outcome, i.e., clinical cure. In some patients with more intractable disease, such as those with limited cerebral softening, scarring after a hemorrhage, glioblastosis, or unrepaired depressed fractures, patients may develop more intractable seizures. For limited lesions, surgical methods can also be considered to control epilepsy, and 70%-80% of patients who are operated on can also become seizure-free. There are also some patients who are difficult to control with both surgery and medications, and neuromodulation therapy and other treatments may be considered. There are also some patients who cannot be completely cured no matter how they are treated. Such patients should have a correct treatment goal and treatment expectation, and generally less than 5%-10% can achieve seizure-free.