IgA nephropathy is the most common primary glomerular disease, which theoretically can not be completely cured, and the current clinical cure standard is: urine protein quantitative less than 0.3g/24h, blood creatinine, urea nitrogen are within the normal range.
The clinical manifestations and pathological changes of IgA nephropathy vary greatly, and the corresponding treatments are also different, which need to be formulated according to the patient’s own situation.
For patients with only microscopic hematuria, the renal function is basically normal and the prognosis is good, usually no special treatment will be given, only need to regular review, avoid overwork and so on.
For patients with obvious proteinuria, ACEI or ARB drugs (such as Benadryl, Valsartan, etc.), hydroxychloroquine, etc. can be used to control urinary protein, and if the effect is not good, glucocorticosteroids such as prednisone acetate and immunosuppressants such as tacrolimus can be added.
Even through these treatments, usually patients will not be completely cured, only need to reach the urine protein quantitative <0.3g/24h, renal function to achieve stability, it has reached the standard of complete remission.
IgA nephropathy patients are recommended to visit regular hospitals and standardize the treatment under the guidance of physicians.