Cardiac muscle is a muscle tissue composed of cardiac muscle cells. The broad definition of cardiac myocytes includes specially differentiated cardiac myocytes that make up the sinus node, intraatrial bundle, atrioventricular junction, atrioventricular bundle (i.e., Heath bundle), and Purkinje fibers, as well as the general working cells of the atrial and ventricular muscles. The first five species make up the pacing and conduction system of the heart, and they contain few or no myogenic fibers, so none of them has a contractile function; however, they are autoregulatory and conductive and are the functional basis for the autoregulatory activity of the heart; the latter two species are contractile and are the functional basis for the diastolic activity of the heart. Dystrophy of myocardial tissue occurs mostly in cardiomyopathies, etc. Caused by cardiomyopathy. Etiology cardiomyopathy cardiomyopathy is a myocardial disease of unknown cause and is generally considered to be related to viral infection, autoimmune reaction, genetics, drug toxicity and metabolic abnormalities. It does not include atopic cardiomyopathies of definite etiology or secondary to systemic diseases. Cardiomyopathies can be divided into three types: dilated cardiomyopathy, hypertrophic cardiomyopathy and restrictive cardiomyopathy. Among them, dilated cardiomyopathy and hypertrophic cardiomyopathy are more common. The cause of cardiomyopathy is still unknown. Dilated cardiomyopathy dilated cardiomyopathy may be associated with certain factors viruses. Myocardial damage due to abnormal metabolism of bacterial drug intoxication is related, of which viral myocarditis is considered to be the most important cause. Damage to myocardial tissue from persistent viral infection, autoimmunity including cellular, autoantibody or cytokine-mediated myocardial injury can cause or induce dilated cardiomyopathy. In addition, multiple factors such as perinatal period, alcoholism, anticancer drugs, disorders of myocardial energy metabolism and abnormal neurohormone receptors can also cause the disease hypertrophic cardiomyopathy may be associated with autosomal dominant inheritance, about 1/3 of them have significant family history, and mutations in cardiac myosin heavy chain and cardiac troponin T genes are the main causative factors. In addition, abnormal catecholamine metabolism, abnormal intracellular calcium regulation, hypertension, and high-intensity exercise can be contributing factors to the pathogenesis of this disease. Restrictive cardiomyopathy is characterized by endomyocardial fibrosis, myocardial stiffness and blocked ventricular diastolic filling. The onset of the disease is slow, and symptoms such as fever, malaise, dizziness and shortness of breath may occur in the early stages, with total heart failure in the later stages. Atrial fibrillation is also common, with some combined with visceral embolism. It is mostly seen in tropical and temperate regions, and there are only disseminated cases in China. Arrhythmogenic right ventricular cardiomyopathy, formerly known as arrhythmogenic right ventricular dysplasia, is characterized by the replacement of the right ventricular myocardium with progressive fibrofatty tissue and is often characterized by right ventricular enlargement, arrhythmias and sudden death. The 1995 WHO/ISFC Expert Committee Report on the Definition and Classification of Cardiomyopathies listed ARVD/C as a Class 4 primary cardiomyopathy along with dilated cardiomyopathy, hypertrophic cardiomyopathy, and restrictive cardiomyopathy. 1. Quit smoking. Even if the nicotine and carbon monoxide content is low, smoking can be harmful to the respiratory organs and coronary arteries. 2, quit eating foods with too much cholesterol. Because high cholesterol in the blood will lead to atherosclerosis and myocardial infarction. 3, quit high blood pressure. Because high blood pressure will increase the burden on the heart and lead to the risk of heart disease. 4. Quit obesity. Because obesity can lead to high blood pressure and increase the level of cholesterol in the blood. 5, quit sitting still. Sedentary and do not participate in physical exercise, the heart has a hundred harm but not a benefit. 6, quit eating food containing too much sugar. High blood sugar, will induce diabetes endanger the heart. 7, abstain from excessive emotional excitement. Because it will disrupt the balance of human organ function and lead to heart disease. 8.Stop drinking too much alcohol. Regular excessive drinking will make the heart rhythm change, and will eventually lead to serious cardiovascular disease. 9, quit living erratically, living in disorder. Long-term, will cause the human organ function disorder, thus increasing the risk of heart disease. 10.Stop being negligent and careless. If you find signs of high blood pressure and high cholesterol, you should seek medical attention and take medication in a timely manner, especially if you have a genetic history of heart disease, you should not take it lightly, because delaying treatment can often lead to irreversible consequences.