Cerebral infarction cannot be cured by taking oral medications alone, but symptoms can be relieved. The general requirement for cerebral infarction to be cured is that the patient is revascularized and free of brain cell necrosis and damage, which is only possible if intravenous thrombolytic therapy with alteplase is administered within the hyperacute time window. Any time this time window is exceeded, the patient is at risk of brain cell necrosis, caused by ischemia and hypoxia. Even after treatment, neuronal regeneration and functional recovery are not possible. So is the follow-up treatment of cerebral infarction equally important? The answer is yes, because there is still an ischemic semidark zone around the patient’s cerebral infarction lesion, and the neurons here are still alive although they have lost their functions, and their functions can be restored after drug treatment, and if the functions of the infarcted area can be compensated, the clinical symptoms of the patient can disappear completely and the standard of clinical cure can be reached, so the follow-up drug treatment of cerebral infarction is still It is very important.