How does chronic obstructive pulmonary disease develop?

I believe that most people are still unfamiliar with slow-onset lung, but they are not unfamiliar with its symptoms such as chronic cough, coughing, shortness of breath or difficulty in whistling. Once you experience similar symptoms, you need to be alert to slow-onset lung. Chronic obstructive pulmonary disease is a multifactorial disease that is centered on inflammation and damage to the lungs. Chronic obstructive pulmonary disease is a stubborn lung disease that can be very harmful to the human body. Many people take it lightly and do not treat it in a timely manner because they do not understand it. Some women, in particular, think that because they don’t smoke, they are less likely to get the disease. In fact, it is too naive to think so. The formation of chronic obstructive pulmonary has a process that is affected by these undesirable factors: First, smoking, it is undeniable that smoking is a major factor causing chronic obstructive pulmonary, because tobacco contains tar, nicotine and hydrocyanic acid and other chemicals, can damage the airway epithelial cells, so that the cilia movement is reduced and macrophage phagocytosis is reduced, thus inducing chronic obstructive pulmonary. Second, prolonged exposure to occupational dust and chemical substances, such as smoke, allergens, industrial exhaust and indoor air pollution, etc., too large a concentration or too long exposure may produce chronic obstructive pulmonary disease unrelated to smoking. Third, air pollution, harmful gases in the atmosphere such as sulfur dioxide, nitrogen dioxide, chlorine, etc. damage the airway mucosa and its cytotoxic effect, so that the cilia clearance function decreases, mucus secretion increases, increasing the conditions for bacterial infection. Fourth, protease and anti-protease to maintain a balance is the main factor to ensure that the normal structure of lung tissue from damage and destruction. Increased protease or insufficient anti-protease can lead to the destruction of tissue structure and produce emphysema. Fifth, infection, infection is one of the important factors in the development of chronic obstructive pulmonary disease. Viruses, bacteria and mycoplasma are important factors in the acute exacerbation of the disease. Sixth, chronic bronchitis, which is the most common cause of chronic obstructive pulmonary disease, due to congenital or acquired impairment of lung bronchial immune function (abnormal mucus secretion, impaired ciliary activity, phagocytic function, inflammatory chemokines, leukotrienes, interleukin 6 and other factors leading to upper whistle infection easily to the bronchi and alveoli development. Seven, chronic recurrent asthma, asthma attacks when the airway spasm, exhaust country difficult, alveolar pressure increased significantly, long chronic obstructive pulmonary emphysema appeared. VIII. Bronchiectasis, with congenital abnormalities of bronchial structure, also related to the pneumonia in early childhood without timely and complete end cure. Nine, the crest lateral deformation: caused by the deformation of the thorax is very easy to produce slow obstructive pulmonary. Therefore, parents who are in a position to do so should pay attention to their children with such congenital developmental abnormalities and perform orthopedic crestoplasty as early as possible to prevent the development of slow-onset lung. The above factors are the factors that lead to the formation of slow-onset lung, and they can occur singly or concurrently.