The diuretic mechanism of furosemide is the effect on water and electrolyte excretion and hemodynamics. 1. On water and electrolyte excretion: it can increase the excretion of water, potassium, calcium, magnesium, phosphorus, etc. Furosemide mainly inhibits the active reabsorption of sodium ions in the thick-walled segment of the medullary collaterals of the renal tubule, so that the difference in the osmotic pressure gradient decreases, and the tubular concentrating function of the renal tubule will be reduced, which will lead to an increase in the excretion of sodium ions. Due to the decrease of sodium ion reabsorption, the distal tubular sodium ion concentration increases, short-term use of the drug can increase the excretion of uric acid, long-term use of the drug may cause hyperuricemia. 2. Effects on hemodynamics: Furosemide can inhibit the activity of prostaglandinolytic enzyme, so that the content increases, thus having the effect of vasodilatation, reducing the resistance of renal blood vessels, thus increasing the renal blood flow as well as the medullary part of the blood supply, and at the same time can also reduce pulmonary congestion in patients with congestive heart failure. Possible adverse reactions to furosemide include thirst, fatigue, and muscle aches. It should be noted that it is contraindicated for those who are allergic to the product. Patients are advised to use the medication under the guidance of a doctor and in strict compliance with the doctor’s instructions, and not to use the medication blindly or change the dosage of the medication without authorization, so as not to cause adverse effects on the body.