High uric acid can lead to a number of complications, especially long-term hyperuricemia. The most common complications are gouty arthritis and gouty nephropathy, which are acute or chronic inflammatory reactions caused by the deposition of uric acid crystals in the joints as well as in the kidneys. Chronic hyperuricemia can lead to an increased incidence of hypertension and coronary heart disease. This is mainly because urate crystals can be deposited on the walls of blood vessels, causing them to become less elastic and leading to an increased incidence of hypertension and atherosclerosis. Chronic hyperuricemia can also cause central nervous system pathology, which generally affects the intracranial vessels as well. Therefore, although hyperuricemia itself is asymptomatic, it presents a series of complications that are very frightening, and once bone destruction occurs as well as chronic renal insufficiency or has caused hypertension, these lesions are irreversible. Only by actively lowering blood uric acid at an early stage can these conditions be prevented.