Snake sores, scientifically called herpes zoster, are caused by reactivation of the varicella-zoster virus, which is latent in the ganglia and is the basis for the development of the disease. The latent virus is activated and replicates along the sensory nerve to reach the skin in the area innervated by that nerve, producing blisters. Simultaneous peripheral and central nerve involvement creates sensory sensitization, resulting in neuropathic pain. The main manifestation is the appearance of clustered blisters in a monochromatic area allocated to the sensory nerve branches of the cranial or spinal nerves, often accompanied by significant neuralgia, and in some patients, systemic symptoms such as malaise and low-grade fever precede the onset of the rash.