Delayed dyskinesia (TD) is an involuntary rhythmic repetitive movement of a group of muscles caused by long-term administration of higher doses of antipsychotics. The incidence varies depending on the type of medication, dose, duration of administration, and individual differences. The main clinical manifestation of tardive dyskinesia is involuntary rhythmic repetitive movements of a muscle group. The common ones are: (1) Mouth-tongue-buccal triad: (2) Involuntary repetitive movements or jerks of the limbs: (3) Trunk muscle movement incoordination: How to prevent it? (1) The focus of this disease is on prevention, and the use of antipsychotics should be clearly indicated. The movement disorders gradually resolve and subside after several months or one to two years of gradual discontinuation of the medication. The need to continue treatment can be replaced with drugs with fewer extrapyramidal side effects, such as clozapine, risperidone (risperidone), olanzapine (olanzepine), quetiapine and so on. (2) Drug treatment There are no effective drugs for TD. Clonazepam is effective in some patients with delayed-onset dyskinesia. (3) Antihistamines such as isoprostanes may be effective. (4) Acute dystonia or acute sedentary inability, diazepam can quickly control the symptoms. The occurrence of this disease is closely related to the long-term use of antipsychotic drugs, should be used reasonably and carefully under the guidance of a doctor. Small doses or short courses of medication are meaningful for the prevention of TD, avoid combining more than two antipsychotics as much as possible, and gradually reduce the dose when discontinuing or replacing antipsychotics, and do not stop them suddenly; those who are old and frail or with organic brain lesions should be given the minimum dose, and anticholinergics should not be used or used sparingly when not necessary.