According to the onset and progression of the disease, it can be divided into the slow progressive type and the acute progressive type, with the slow progressive type being the most common. Early manifestations: Early stage is asymptomatic, but occasionally the blood pressure is found to increase during physical examination, or dizziness, headache, blurred vision, tinnitus, insomnia, fatigue, lack of concentration and other symptoms after nervousness, emotional excitement or exertion, which may be caused by senior mental dysfunction, early blood pressure is only temporarily elevated, with the progress of the disease blood pressure continues to rise, organ involvement. 2. Brain manifestations: headache and dizziness are common, mostly triggered by emotional excitement, excessive fatigue, climate change or discontinuation of antihypertensive drugs, sudden increase in blood pressure, severe headache, visual impairment, nausea, vomiting, convulsions, coma, transient hemiparesis, aphasia, etc. 3.Heart manifestation: In the early stage, the heart function is compensated and the symptoms are not obvious; in the later stage, the heart function is not compensated and heart failure occurs. 4, renal manifestations: long-term hypertension caused by small renal artery sclerosis, renal hypofunction, can cause nocturia, polyuria, urine containing protein, tubular and red blood cells, low urine concentration function, phenol red excretion and urea contouring disorders, azotemia and uremia. 5.Arterial changes. 6, fundus changes. Second, acute hypertension, also known as malignant hypertension, accounting for 1% of hypertension, can be suddenly transformed from the slowly progressive type, can also start, malignant hypertension can occur at any age, but the most common 30-40 years old, blood pressure is significantly elevated, diastolic blood pressure is more than 17.3Kpa (130mmHg), there is weakness, thirst, polyuria and other symptoms, rapid vision loss, retinal hemorrhage and fundus. There is rapid loss of vision, retinal hemorrhage and exudation in the fundus, often bilateral optic nerve papillary edema, rapid development of proteinuria, hematuria and renal insufficiency, heart failure, hypertensive encephalopathy and hypertensive crisis can also occur, and the disease progresses rapidly and most die from uremia.