During neurology consultations, we often encounter people who suffer from cerebral infarction at a young age and are unable to take care of themselves, which seriously affects their quality of life. By asking the medical history, most of them suffer from diabetes, some of them have diabetes but do not actively control blood sugar, and some of them are found to have diabetes when the blood sugar is checked at the time of cerebral infarction, and these conditions can be avoided if the blood sugar is actively controlled. Diabetes is one of the main causes of cerebrovascular lesions, and the risk of cerebrovascular disease in diabetic patients is 4 to 10 times higher than that in non-diabetic patients, 85% of whom have cerebral infarction. More than 30% of patients with acute cerebral infarction are accompanied by hyperglycemia, 15% of whom have been diagnosed with diabetes before the onset of the disease, and 15% of whom do not even know they have diabetes at the time of cerebral infarction. Cerebrovascular accidents can lead to disability or death in diabetic patients. Diabetic cerebrovascular lesions are prone to factors such as hyperglycemia, hyperinsulinemia, hypertension, hyperlipidemia, and hypercoagulability, among which hyperglycemia can increase blood viscosity, increase platelet adhesion and aggregation, and vascular endothelial cell damage. These changes can promote thrombosis and cause cerebral infarction. Diabetic cerebrovascular accidents are characterized by multiple, large cerebral infarcts that are common and prone to diabetic hyperosmolar coma (an acute complication of diabetes). 10% to 25% are lacunar cerebral infarcts, which usually do not have limb paralysis, but only show headache, dizziness, memory loss, unresponsiveness, limb numbness, and ataxia, and are easily missed. At the onset of cerebral infarction, the higher the patient’s blood sugar, the worse the healing process. The death rate of diabetic cerebral infarction is four times higher than that of non-diabetic patients. The prevention and treatment of diabetic cerebrovascular disease is basically the same as that of non-diabetic patients, but it should be more active and the measures should be more effective. Diabetes must be detected early and controlled effectively to delay the onset and development of diabetic cerebrovascular disease. Effectively lower blood pressure, adjust blood lipids, abstain from alcohol, lose weight, eat a low-salt, low-fat diet, and exercise appropriately. Vasoactive drugs and thrombolytic drugs can be taken under the guidance of doctors to reduce blood viscosity. If symptoms such as sudden dizziness, headache, dizziness, nausea, numbness, blurred vision and movement loss occur, send to hospital immediately to get proper diagnosis and treatment in time to reduce the harm caused by stroke.