Although most cases of patent foramen ovale close spontaneously before the child is one year old, some patients with patent foramen ovale require surgical treatment. In particular, recent studies have shown a close relationship between patent foramen ovale and cardiovascular diseases such as migraine in adults. Modern medicine has also shifted from traditional surgical treatment to interventional surgical treatment. Interventional treatment of patent foramen ovale has become one of the main treatment modalities for patent foramen ovale. Usually, when patients choose interventional treatment, they have the question of whether the interventional blocker will fall off. The fear is that the interventional blocker will fall off and cause repeated pain to the patient. First of all, let’s talk about the pathogenesis of patent foramen ovale. The foramen ovale is a vital channel necessary for the development of the fetus, and it is through this channel that blood from the mother’s umbilical vein enters the left side of the fetal heart and is then distributed throughout the body to provide the oxygen and nutrients needed for fetal development. At birth, with the first cry, the pressure in the left atrium rises and the oval fossa valve is pressed against the edge of the oval fossa to form a functional closure, which is usually complete anatomically until 5 to 7 months after birth. Therefore, it is possible that the foramen ovale may remain open up to one year of age, and there may be a small shunt, or even 5%D10 % of individuals with the foramen ovale remaining open and unclosed for life, but with no effect on cardiac hemodynamics. Therefore, in infants, the patent foramen ovale is a normal physiological phenomenon, not a precordial disease, and usually does not require surgery. However, if the foramen ovale opacification defect is large, greater than 8D10 mm, with a high fractional flow, care is needed. The physiological principle of patent foramen ovale is that a heart with a local anatomical abnormality caused by a disorder in the formation of the heart and great vessels during human embryonic development (within the first 2-3 months of pregnancy), or a failure to close a channel that should close automatically after birth (which is normal in the fetus), is called congenital heart disease. Most cases of patent foramen ovale close spontaneously before the child is one year old and do not require surgical treatment. However, some patients do require treatment, especially since recent studies have found a close association between adult patent foramen ovale and related cardiovascular diseases such as migraine headaches, so treatment such as interventional surgery is the best way to heal patent foramen ovale. In general, children over one year old with patent foramen ovale can be considered for interventional treatment if they do not have pneumonia or bronchitis and are able to tolerate it well, and it is a one-time treatment and a complete cure. It is the treatment of choice for children with patent foramen ovale, as it is less painful, has no surgical scar, and is safe and reliable. At present, open-heart surgery is not necessary for oval foramen insufficiency. At the same time, interventional closure should be done in a tertiary hospital or a hospital with strong expertise. After the interventional closure of the patent foramen ovale, after a few months, the body tissue will be covered with the encapsulated blocker, and the original defective patent foramen ovale will be repaired intact, and the patent foramen ovale can be completely cured. After the interventional occlusion of the patent foramen ovale has completely healed, at this time, the occluder will gradually become firmly integrated with the patient’s tissue over time, thus not causing repeated occlusion of the patent foramen ovale. By the same token, other precordial diseases such as patent ductus arteriosus, ventricular septal defect, atrial septal defect, etc. are completely cured after successful blocking of the patient, no matter how the child grows up, the blocker is already firmly bonded to the patient’s tissue to form one, and it is impossible for the blocker to fall off as the patient grows older and the organ grows up. After the cure, the patient can live, study and work like normal people.