Luminal cerebral infarction is a cerebral infarction caused by occlusion of a deep penetrating artery in the brain. The diameter of the lesioned vessel is mostly 100~400um, and the diameter of the infarct foci left by macrophagy is generally 0.2~15mm, which is called luminal cerebral infarction. Clinical symptoms are related to the size and location of the luminal infarct foci, and the following types are common: 1. Pure motor stroke: manifested as paralysis of the face, tongue, and limbs to varying degrees without sensory impairment, visual field loss, aphasia, etc. The lesions are located in the radial crown, internal capsule, basal ganglia, cerebral bridge, and medulla oblongata. 2. Pure sensory stroke: The patient complains of hemianesthesia, and examination reveals hyperalgesia or loss of sensation in one limb or body. The lesion is in the contralateral posterior lateral nucleus of the ventral thalamus. 3, Ataxic mild hemiparesis: manifests as pure motor mild hemiparesis and cerebellar ataxia on the contralateral side of the lesion, with the lower extremities being the most important, also with dysarthria and nystagmus. Corticobasal tract at the internal capsule or at the base of the cerebral bridge. 4. Sensorimotor stroke: Mostly with hemianesthesia followed by mild hemiparesis. The posterior ventral nucleus of the thalamus and involves the posterior limb of the internal capsule. 5. Dysarthria hand clumsiness syndrome: The patient has severe dysarthria, dysphagia, central facial tongue palsy on the contralateral side of the lesion, mild weakness of the hand on that side, inaccurate finger-nose test, unsteady gait, hyperactive tendon reflexes and positive pathological reflexes. The lesion was located at the base of the pontine brain or in the forelimb and knee of the internal capsule.