Objective: To explore the clinical manifestations, diagnosis and treatment methods of rhinogenital orbital herniation. Methods: We retrospectively analyzed the clinical manifestations, diagnosis, treatment and surgical access of 89 cases of sinus tumors invading the orbit. Results: Of the 89 cases, 11 cases had orbital periosteum destruction; 78 cases had intact orbital periosteum, and the orbital periosteum moved when compressed on the affected side of the eyeball, and the protrusion of the eyeball slightly improved on the day of the operation, with diplopia, and the protrusion of the eyeball and diplopia disappeared gradually in 2-3 weeks. There was no cerebrospinal fluid leakage, no postoperative cranial nerve symptoms, and no surgical complications. 89 cases were followed up in outpatient clinics for 6-24 months, with epithelialization of the sinus cavity, good open drainage, and no recurrence. No neoplastic organisms were seen in the nasal cavity of 89 cases at 6 months postoperatively, 7 cases had nasal adhesions, 1 patient with malignant tumor developed cataract after 1 year due to radiotherapy, 2 cases of malignant tumor had no recurrence at 3 years of follow up, 1 case recurred after 2 years, and 3 cases died after 3 years. DISCUSSION: Proptosis of rhinogenic origin, a relatively common clinical syndrome, can be caused by sinus tumor or cyst invasion into the orbit, and the tumor develops toward the orbit causing the eyeball to protrude forward, which is often misdiagnosed as a primary tumor in the orbit. The position of the eyeball caused by sinus tumor can be different due to different sinuses. Maxillary sinus lesions make the eyeball protrude forward and upward, and the downward rotation of the eyeball is limited, accompanied by conjunctival edema; lesions originating from the anterior group of sieve sinuses often make the eyeball shift outwardly or externally, and it is difficult to rotate the eyeball internally; lesions originating from the frontal sinus make the eyeball shift outwardly and downwardly, and the upward rotation of the eyeball is limited; lesions of the pterygoid sinus and the posterior group of sieve sinus make the eyeball protrude forwardly without deviation, but it can be caused by the tumor or cysts. The lesions in the posterior group of the pterygoid sinus and sieve sinus usually cause the eyeball to protrude to the front without deviation, but the damage to the optic nerve may result in signs of optic disk edema and optic nerve inflammation or atrophy. The sinus tumors may also be paralyzed by the extraocular muscles, which may result in strabismus and eye movement disorders.CT scan and coronal scan can clearly show the damage of the bone wall and the structural relationship between the tumor and the orbits and sinuses, and MRI can show the overall morphology of the tumors. In this group, several patients with huge mucus cysts in maxillary sinus, sieve sinus and orbit were quickly diagnosed by CT, MRI and nasal endoscopy, and surgical methods and approaches were correctly selected to avoid blind orbital opening. Combined with the cases in this group, we believe that: nasogenic intraorbital tumors are mostly located in the infraorbital, infraorbital and supraorbital regions, and the surgical approach can be made through the oral cavity and nasal cavity, so the orbital cavity should not be opened, and if the orbital cavity needs to be opened, it is best to select the three nearest parts of the orbital cavity, infraorbital and supraorbital regions. If there is difficulty in the transnasal and oral approaches, then the orbit should be opened jointly. The orbital harassment should be avoided as much as possible. This reduces facial scarring and establishes drainage in the nasal tract. Rapid intraoperative sectioning of malignant tumors seeks to completely remove diseased tissue postoperatively supplemented with radiation and chemotherapy.