Cavernous infarction in the left lateral ventricle is formed due to ischemia and occlusion of the deep perforating artery supplying the small lateral ventricle, resulting in ischemia, hypoxia, and necrosis of the local brain tissue. If the patient is found to be asymptomatic cavernous infarction during physical examination, and if the patient shows weakness of the right limb or numbness of the limb, it may be blocking the thalamus or the internal capsule and other functional areas, which should be actively treated. In the acute stage, patients can be given thrombolytic drugs, such as the use of rt-PA treatment. If there is no time window for thrombolysis, we should consider using aspirin and clopidogrel to fight platelet aggregation, and try to improve the blood supply to the brain and neurological function of the patient.