The most typical symptom of left renal vein compression syndrome is the presence of hematuria, which is mainly discharged from the left ureter. The main pathogenesis of left renal vein compression syndrome is that the left renal vein enters the vena cava, which is pressed by the abdominal aorta and superior mesenteric artery, and the resistance to blood flowing back into the vena cava becomes greater, leading to the compression of the left renal vein, which will result in hematuria in the renal pelvis, and many people can have significant relief through bed rest after hematuria occurs. The easiest way to treat this is to increase the fat content, make the pinch of compression larger, and reduce the compression, which may require stent placement if necessary.