It is currently believed that atherosclerotic plaques are formed by lipid deposition after damage to the arterial intima, so a simple increase in blood lipids does not necessarily lead to plaque formation. Risk factors for atherosclerotic plaque formation include hypertension, smoking, age (> middle-aged and elderly people over 40 years old), diabetes mellitus and glucose tolerance abnormality, etc., and dyslipidemia is only one of them. The formation of atherosclerotic plaques is in a variety of risk factors, lipid deposition through the damaged endothelium in the inner wall of the blood vessels to form lipid streaks, and gradually evolve into fibro-fatty lesions as well as fibrosclerotic plaques. However, dyslipidemia, i.e., hyperlipidemia, is the most important risk factor for atherosclerosis, and therefore people with hyperlipidemia need to be treated aggressively to reduce the likelihood of atherosclerotic plaque formation.