High phosphorus causes acute renal failure, hypocalcemia, and increases mortality from vascular diseases, among other things. Inorganic phosphorus is measured in the serum, and hyperphosphatemia is defined as a blood phosphorus concentration >1.45 mmol/L. High phosphorus by itself does not produce specific clinical symptoms. Acute hyperphosphatemia, however, increases the risk of calcium and phosphorus deposition, which can lead to metastatic calcification of soft tissues and may cause acute renal failure. Decreased blood calcium can cause convulsions, hypotension, and cardiac arrhythmias. Chronic hyperphosphatemia due to chronic kidney disease increases mortality if serum phosphorus concentration is higher than 2.08 mmol/L. Hyperphosphatemia is also a risk factor for coronary artery disease and other vascular calcifications and is associated with increased mortality. In addition to the above, hyperphosphatemia has other hazards, such as induced epilepsy, etc. It is recommended to go to the hospital for further examination and timely compliance with the doctor’s instructions for targeted treatment, so as not to delay the course of the disease.