Cerebral infarction is a blockage of cerebral blood vessels, which can be divided into atherosclerotic thrombotic cerebral infarction, lacunar cerebral infarction, cerebral watershed infarction, and cerebral embolism depending on the cause. The main etiologies of atherosclerotic thrombotic cerebral infarction include hypertension, hyperlipidemia, hyperglycemia and hyperhomocysteinemia. Patients may suffer from ischemic necrosis or softening of the brain due to narrowing of the lumen and stiffening of the walls due to atherosclerosis, which leads to ischemia and hypoxia in the brain due to blockage of the blood vessels. If the patient is a large cerebral infarction, he or she can develop sensory-motor impairment of the lateral limbs, as well as impaired consciousness, which can lead to death in severe cases. If the patient is a cerebral embolism, most of them are caused by the dislodgement of emboli of cardiac origin blocking the blood vessels or the dislodgement of thrombus emboli of large arteries, blocking the blood vessels. If the patient is a lacunar cerebral infarction, most of them are caused by occlusion of small blood vessels at the end due to long-term hypertension, and if the patient is a cerebral watershed infarction, most of them are caused by hypoperfusion.