Tenosynovitis is a chronic aseptic inflammatory change of the tendon sheath caused by mechanical friction. In daily life and work, due to frequent activities caused by excessive friction, coupled with some parts of the bony bulge or tendon travel direction to change the travel angle, which increases the mechanical friction of the tendon and tendon sheath, this mechanical stimulation, can make the tendon sheath in the early stage of congestion, edema, exudation and other sterile inflammatory reaction. After repeated trauma or prolonged period of time, chronic fibrous connective tissue hyperplasia, hypertrophy, adhesion and other changes occur, and the thickness of the tendon sheath can be thickened from 2.5px to 0.2-7.5px, resulting in tendon sheath stenosis due to the thickening of the tendon sheath. Adhesions between the tendon sheath and the tendon may occur to different degrees, and the tendon may become degenerate and deformed, showing a thickened gourd shape at both ends or a thickened tissue hyperplasia at the damaged area to form an enlarged middle and thin fusiform shape at both ends. The clinical manifestations are local pain, pressure pain and limitation of joint movement, etc. The common sites are the beginning of the fibrous sheath of the flexor muscles of the fingers or thumbs, and the tendon sheath of the short extensor muscles of the thumb at the radial tuberosity. Radial stenosing tenosynovitis, also known as de Quervain’s disease, is a tenosynovitis of the sheaths of the short extensor thumb tendon and the long extensor thumb tendon. It occurs mostly in women between 30 and 50 years of age. Treatment options are conservative and surgical. Conservative treatment includes: braking of the affected limb, physical therapy, and closure. For those who cannot be relieved by non-surgical treatment or who have long and recurring disease, and whose appearance is significantly affected by the protruding bulge of the radial styloid process, surgical treatment can be considered.