Chronic glomerulonephritis can be treated by actively controlling high blood pressure and reducing urinary protein, limiting the amount of protein and phosphorus in food, taking medication and avoiding factors that aggravate kidney damage such as infection. 1. Actively controlling high blood pressure and reducing urinary protein: beta-blockers such as metoprolol tartrate, angiotensin-converting enzyme inhibitors such as captopril, angiotensin receptor antagonists such as valsartan and other drugs can be taken. 2. Limit the intake of protein and phosphorus in food: the intake of protein and phosphorus should be limited, and according to the condition of renal function, a high-quality low-protein diet such as fish and chicken should be given, and the intake of phosphorus in diet should be controlled at the same time. 3. Take glucocorticoid and cytotoxic drugs: If the patient’s renal function is normal or only mildly impaired, and the pathological type is mild (e.g. mild tethered proliferative nephritis, early membranous nephropathy, etc.), and the urine protein is large, and those who do not have contraindications can take glucocorticoid, such as methylprednisolone, prednisone acetate, etc., and cytotoxic drugs, such as cyclophosphamide and tacrolimus, to improve the symptoms. 4. Avoid aggravating factors of renal damage: infection, exertion, pregnancy and nephrotoxic drugs such as aminoglycoside antibiotics can damage the kidneys and lead to deterioration of renal function, which should be avoided. It is recommended that patients with chronic glomerulonephritis consult a doctor in time and take medication as prescribed by the doctor to avoid aggravating their condition.