In general, infants born with low birth weight (less than 2500g) with congenital anomalies (including all causes of brain developmental abnormalities; 53% of cerebral palsy patients with tetraplegia are associated with congenital anomalies; 35% of cerebral palsy patients with non-tetraplegia are due to congenital dysplasia) are at extremely high risk of cerebral palsy. Of course, there are many other possible clinical causes of pediatric cerebral palsy, such as cerebral ischemia and hypoxia. Among cerebral palsy patients, 20% are caused by asphyxia and birth injury. Factors leading to ischemia and hypoxia can be divided into the following: 1. Maternal factors: such as suffering from gestational hypertension syndrome, heart failure, hemorrhage, anemia, shock or drug addiction, drug overdose, etc.; 2. Placental factors: such as placental abruption, placenta praevia, placental necrosis or placental malfunction, etc.; 3. Umbilical cord blood flow blockage: such as cord prolapse, compression 4, abnormal delivery process: such as breech delivery, stalled delivery, surgical delivery (forceps) or the application of anesthetics; 5, neonatal factors: in addition to asphyxia, there are many abnormal cardiopulmonary diseases. For example, congenital heart disease, respiratory distress syndrome, peripheral circulatory failure, erythrocytosis In addition, nuclear jaundice is also one of the important causes of pediatric cerebral palsy, with the progress of medicine, the proportion of cerebral palsy caused by nuclear jaundice has decreased. We know that there is a so-called pathological jaundice in newborns, and one of the pathological jaundices is very dangerous, i.e. the jaundice occurs too early and deepens rapidly. This is caused by an influx of bilirubin into the blood. Some of the bilirubin that enters the blood is “free” bilirubin (medically known as free bilirubin), which is not bound to protein. Since no protein is bound to it, it crosses the blood vessel wall and enters the brain tissue to stain the nuclei of central nerve cells yellow, hence the name “nuclear jaundice. After the nerve cells are stained, they are unable to metabolize energy, and degeneration and necrosis occur. Therefore, when a child has cerebral palsy, the symptoms are mainly neurological damage. In the early stages, there is mental depression, drowsiness, weakness in sucking milk, followed by non-feeding (milk refusal), moaning, scream-like crying, and eye inactivity (staring); if it deepens further, there is head tilting backwards, corneal inversion, and convulsions. Although pediatric cerebral palsy is a lifelong disability, the condition can be effectively improved if the child receives timely scientific and effective rehabilitation treatment. Parents should pay close attention to their children’s growth and development, and once any abnormality is detected, do not delay and make sure to get treatment and training. Research has confirmed that before 6 months of age is the critical period for the treatment of children with cerebral palsy. The early brain tissue of infants is in a period of vigorous growth and development, and early intervention at this time is effective, and the movement disorder is easier to recover after treatment. Once a child is diagnosed, early treatment is the key to their future mobility and quality of life. Currently, the main treatment for cerebral palsy is rehabilitation and surgery. Rehabilitation training mainly includes physiotherapy, acupuncture, massage, limb exercise, and equipment training. There are various surgical treatment methods and procedures, such as functional selective posterior spinal nerve root dissection (FSPR), carotid epicraniectomy, peripheral nerve surgery, orthopedic surgery, etc. It is important to remember that orthopedic surgery should be performed after decompression surgery, otherwise the results are not stable. For children with spastic cerebral palsy with high muscle tone and abnormal gait, FSPR can be performed to adjust the muscle tone of the spastic muscles as close to normal as possible by treating the posterior spinal nerve roots. After the FSPR procedure, orthopedic surgery and rehabilitation training can be performed according to the specific conditions of the child, and the results are very significant. After the surgery and postoperative follow-up, most of the children showed different degrees of improvement in both limb and intelligence. On the one hand, the child’s muscle tone was significantly reduced, the flexibility and fine activities of the limbs were increased, and the coordination was improved. At present, the scientific treatment method for cerebral palsy recognized by the academic community is: rehabilitation-surgery-orthopedic-rehabilitation, so as to achieve the best rehabilitation effect. Our clinical practice over the years has confirmed that the treatment of pediatric cerebral palsy is a long-term process, and it is impossible to rely on a single surgery or a short-term rehabilitation training to achieve immediate results. Children with cerebral palsy who are eligible for surgery should receive surgery at the best time (between the ages of 2.5 and 6 years old), and they should adhere to long-term formal rehabilitation training before and after surgery in order to ensure the rehabilitation effect.