Lumbar disc herniation is one of the common orthopedic diseases, and about 1/5 of the patients with low back pain are caused by lumbar disc herniation. It has been more than seventy years since Mixterher and Barr proposed this disease in 1934. From the epidemiological analysis at home and abroad, the population rate and absolute value of its incidence are on the rise. The age of onset varies from a few years to tens of years, and we have seen patients with lumbar disc prolapse as young as 9 years old. The increase in the incidence of this disease is related to the environment we live in and the change in our living and working habits. Long-term bad habits with the back are the main cause. In the past seventy years, the majority of medical workers have been working hard on research and have made great progress both in the understanding of the disease and in the treatment methods. From folk recipes, herbal medicine, to traction, massage, physiotherapy, reset to interventional, minimally invasive, surgical treatment, treatment methods are endless and varied. How can we choose among the many treatment methods for individual patients? Patients are headless, and many doctors do not know, full of confusion. In fact, the choice of indications is the key to treating the disease. That is, in a sea of treatment methods, understanding the extent of your condition and choosing the most targeted treatment method, lumbar disc herniation can be cured completely and quickly. The actual fact is that you can find a lot of people who have been in the business for many years. The actual fact that I’m not very knowledgeable, especially compared to the authoritative experts at home and abroad, the comments that are suitable for the following consultation are just for reference and not final. The pathology of lumbar disc herniation The intervertebral disc tissue itself lacks blood supply and has very poor repair ability, coupled with a lot of weight-bearing activities. Generally, after the age of 20, the intervertebral disc begins to undergo degenerative changes, and the toughness and elasticity of the fibrous ring are gradually decreasing. At this time, if trauma is encountered, especially cumulative strain injury, it becomes the cause of rupture of the annulus fibrosus. There are also many cases where there is no history of trauma, but where the tension in the muscles and ligaments increases after a cold, which increases the internal pressure of the disc and promotes the rupture of the atrophied annulus fibrosus. The intervertebral disc is a special structure made of connective tissue that is burdened with unique functions. Any alteration of the disc affects its normal mechanical performance or interferes with its normal balance function, absorption and redistribution of its forces to the spine. The intervertebral disc includes the nucleus pulposus, the annulus fibrosus, and the cartilage plate. The nucleus pulposus of the intervertebral disc contains a small amount of collagen fibers in addition to a soft matrix of mucopolysaccharide-based fibrils. The nucleus pulposus accounts for more than half of the volume of the intervertebral disc and, because of its deformability, is able to transmit load forces appropriately. The ability of the intervertebral disc to maintain proper function is closely related to its water content water content, which in turn is stabilized by the polysaccharide content. Although the difference between the fibrous annulus and the nucleus pulposus is still significant, the collagen fibers of the fibrous annulus are in the form of dense laminae, with the fibers of each lamina staggered at right angles to each other and at an angle of 45° to the spine, and this laminae structure accommodates the pressure and tension and the flexion and rotational stresses caused by the spine. The cartilage plate is glassy cartilage, which is scarfed between the vascularized vertebral spongiosa and the avascular nucleus pulposus. On the surface of the vitreous cartilage, the collagen fibers are parallel to each other on the surface, and at the deeper level near the bone, the collagen fibers are perpendicular. The proteoglycan ground intervertebral disc matrix is an important component of the intervertebral disc matrix and is an important structure for the mechanical and chemical functions of the disc. Proteoglycan molecules are large, extremely viscous, and very hydrophilic. Under normal conditions, the nucleus pulposus has a strong compressibility and, due to the properties of proteoglycans, it has a strong loading capacity. If the glycan chains of the proteoglycans break down, they lose their ability to retain extracellular water. The biochemical integrity of the intervertebral amidships nucleus pulposus is determined by its water-bearing volume. Under normal conditions the intervertebral discs bear pressure and redistribute their forces to the spine and are an essential part of accomplishing normal function. The formation of disc herniation is a normal excess of proteoglycans, which will cause fluid and increase in the nucleus pulposus, and the pressure within the nucleus pulposus rises and predisposes to disc herniation. However, mucopolysaccharides in the nucleus pulposus can produce a new balance through reduction and re-integration. The progressive reduction of protein polysaccharide can promote the fibrosis of collagen, and the nucleus pulposus gradually loses its original compressibility and loading capacity due to the deposition of collagen and the increase of fibrosis, which will not be able to perform the function of absorbing and redistributing the stress to the spine by the nucleus pulposus of the intervertebral disc under load at any time, thus causing damage to the intervertebral disc. If external trauma or excessive stress is applied to the damaged disc, it is more likely to cause disc herniation. It is believed that the glycoprotein and β-protein in the matrix of the nucleus pulposus form an antigen in the case of autoimmunity, and it is this antigen that is released (referring to the release of β-protein in degenerated discs and herniated discs, which is normally encapsulated in the nucleus pulposus), which continuously stimulates the body and thus generates an immune response, and also causes an inflammatory response in the nerves, resulting in pain.