As a professional otorhinolaryngologist, we have heard very little about Empty Nose Syndrome, which has been a raging issue in otolaryngology for some time. The term “Empty Nose Syndrome”, first written by Eugene and Stenkvis in 1994, is used to describe the absence of nasal tissue and the absence of normal anatomy of the nasal cavity on imaging. The term “empty nose syndrome” was first coined by Eugene and Stenkvis in 1994 to describe the absence of nasal tissue and certain symptoms associated with the absence of normal nasal anatomy on imaging. Whereas in the past empty nose syndrome was misdiagnosed as atrophic rhinitis, there is now an improved understanding of the disease and, importantly, a distinction between this disease and primary atrophic rhinitis, as well as a new understanding of the clinical presentation, diagnosis and treatment of this disease. The etiology of this disease is clear: excessive turbinate resection surgery can lead to secondary nasal mucosal atrophy and a series of accompanying symptoms. Clinical manifestations are nasal congestion and dryness of the nasal cavity and/or nasopharynx and pharynx, with some patients experiencing a sense of suffocation, inability to concentrate, fatigue, irritability, anxiety, depression, thick nasal discharge, bloody discharge, bad odor, and decreased sense of smell. Nasal examination reveals a spacious nasal cavity in the shape of a “tube”. The nasal endoscopy shows a wide nasal cavity with dry, pale mucous membrane, occasional crusting, and the presence of one or two absent turbinate tissues. The diagnosis of empty nose syndrome is based on: 1. medical history, history of previous turbinate removal surgery. 2. clinical symptoms, including at least: nasal congestion and dryness of the nasal cavity and/or nasopharynx and pharynx, some patients have a combination of nasal crusting, nasal pus, bad odor, bloody nasal discharge, mental depression, etc. 3. rhinoscopic examination, the mucosa of the nasal cavity has varying degrees of atrophy, dryness or crusting, normal turbinate structure is absent, the nasal cavity is barrel-shaped Enlarged, can look directly into the nasopharynx. This needs to be differentiated from atrophic rhinitis. Conservative treatment of the disease is effective, but the outcome is poor. The prevailing treatment option is to narrow the overly spacious nasal cavity. The exact site, degree of narrowing, and materials used vary from person to person. Submucosal filling of the inferior turbinate with tipped rectus abdominis fascia rib cartilage pieces is effective in improving symptoms in patients with hollow nose syndrome. Reduction of the anterior nasal aperture can significantly alleviate or even cure the “empty nose syndrome”. Prevention of this disease is the main focus of prevention. Strict control of the extent of turbinate removal, selection of the appropriate inferior turbinate surgery, and attention to the protection of the nasal mucosa during endoscopic surgery are the main means of prevention of empty nasal syndrome.