Left ventricular hypertrophy refers to hypertrophy and interstitial hyperplasia of left ventricular wall and septal cardiomyocytes arising from various complex pathophysiological mechanisms. Left ventricular hypertrophy occurs in hypertension for two main reasons: 1. Catecholamines and growth factors such as angiotensin II stimulate cardiomyocyte hypertrophy and interstitial fibrosis. 2, hypertension causes an increased pressure load, left ventricular hypertrophy is one of the compensatory processes for increased left ventricular myocardial tone; the occurrence of left ventricular hypertrophy capillary bed can not be correspondingly increased will cause myocardial cells ischemia and hypoxia. Left ventricular hypertrophy needs to be diagnosed by electrocardiogram, X-ray chest film and echocardiogram, among which echocardiogram has the highest sensitivity. Left ventricular hypertrophy directly causes cardiac decompensation and accelerates the process of coronary atherosclerosis, which results in increased myocardial ischemia, i.e., coronary heart disease; it leads to the occurrence and development of arrhythmias, sudden death and heart failure. Left ventricular hypertrophy is an independent risk factor for cardiovascular disease prevalence and mortality. Clinical studies have shown that left ventricular hypertrophy and its secondary manifestations are closely associated with cardiovascular events. Left ventricular hypertrophy alone can be completely asymptomatic, but left ventricular hypertrophy predisposes to coronary atherosclerotic heart disease and heart failure, when chest tightness and chest pain, palpitations and shortness of breath after activity, breath-holding at night and even inability to lie flat and swelling of the lower extremities occur. The primary treatment for hypertensive patients with left ventricular hypertrophy is still blood pressure control: the best choice of antihypertensive drugs is to choose long-acting preparations, so that the 24-hour blood pressure is stabilized at a more constant and near-normal level; the choice of antihypertensive drugs is the strongest effect of angiotensin-converting enzyme inhibitors to reverse left ventricular hypertrophy, followed by calcium antagonists, beta-blockers, etc.; those with abnormally high blood viscosity should also The high sodium intake can promote myocardial hypertrophy, so limiting the sodium intake can reduce the weight of the left ventricle.