Bile reflux gastritis a special type of chronic gastritis, commonly after gastrectomy and gastrointestinal anastomosis, the total incidence is about 5%, where the incidence after Billroth II type of gastrectomy is 2 to 3 times higher than that of Billroth I type. Under normal physiological conditions, duodenogastric reflux exists in the organism, and the reflux does not cause damage to the gastric mucosa. However, in patients with bile reflux gastritis, due to impaired gastric-pyloric-duodenal motility, duodenal contents (such as bile acids and bile salts) reflux into the stomach and, under the action of gastric acid, destroy the gastric mucosal barrier, causing H+ reperfusion into the epithelium, resulting in chronic inflammation, erosion and even ulceration of the gastric mucosa, followed by a series of manifestations such as epigastric pain, vomiting of bile, bloating and weight loss. Biliary reflux gastritis can be divided into primary biliary reflux gastritis and secondary biliary reflux gastritis: the former is non-surgical gastric occurring in excess duodenal fluid reflux; the latter is gastric bile reflux occurring after gastric pylorus surgery or gallbladder removal. Long-term bile reflux can lead to esophagitis, gastric mucosal erosive, proliferative, active inflammation, gastric ulcer, and even contribute to the occurrence of gastric cancer. Wei Zhi, Department of Gastroenterology, General Hospital of Jinan Military Region