Biliary reflux gastritis is a specific type of chronic gastritis caused by the reflux of bile from the duodenum into the stomach. The bile refluxes into the gastric sinus, which is closest to the pyloric opening (the place where food passes through to connect the stomach to the duodenum), causing damage to the mucosa there. The main symptoms of the disease are a feeling of fullness or discomfort in the upper abdomen, with vague or severe pain, often in periodic episodes, and may be accompanied by bloating, belching (burping), acid reflux, heartburn, nausea, vomiting, loss of appetite and weight loss. Gastroscopy shows that bile is constantly pouring into the stomach from the pylorus, and the gastric mucosa (especially the mucosa of the gastric sinus) is obviously edematous, congested, rough, and bleeds easily when touched, with a dirty surface and yellow-green bile. The cause of this disease: Under normal circumstances, the pyloric opening is contracted and closed. When the food in the stomach is discharged into the duodenum by gastric peristalsis, the pyloric opening is diastolic and open. In the following cases, after partial gastrectomy, vagotomy, pyloroplasty, gastrointestinal anastomosis, gallbladder removal, congenital incomplete closure of the pyloric valve, etc., the “gatekeeper” role of the pyloric valve is destroyed, and bile reflux can occur. The incidence of this disease increases after cholecystectomy (60% of patients with this disease have a history of cholecystectomy), which is related to the uninterrupted flow of bile into the duodenum 24 hours a day (in a normal gallbladder, bile enters the duodenum regularly after meals). In case of bile reflux (to be precise, reflux of duodenal fluid mixed with bile), bile, pancreatic enzymes and lecithin in duodenal fluid can break the barrier effect of gastric mucosa. Bile reflux gastritis prevention and control measures: 1, oral gastric power drugs such drugs can inhibit the reflux of bile into the stomach, commonly used: morpholine (multipanolide), new nona (mosapride). Can enhance gastrointestinal peristalsis, regulate the normal activities of the gastrointestinal tract, so that food from the stomach into the small intestine, and inhibit bile reflux, generally 15 to 30 minutes before meals to take. The duration of administration should not be less than 2 weeks. 2, oral gastric mucosal protective agent commonly used drugs are: ① thioglycollate: can be complexed with the mucin of the gastric mucosa to form a protective film to protect the gastric mucosa from bile damage; ② Similac: gastric mucosal protective agent, has the effect of strengthening the gastrointestinal mucosal barrier, conducive to the regeneration of the gastric mucosa. 3, diet therapy diet should be light, do not eat greasy food, so as not to stimulate increased bile secretion, aggravate reflux and the condition. The actual fact is that you will be able to get a lot more than just a couple of hours of work. Avoid drinking strong tea, strong wine, strong coffee and eating spicy, too cold, too hot and rough food. 4. Remove certain aggravating factors including smoking cessation, avoiding emotional stress and not taking drugs that irritate the gastric mucosa, such as aspirin, anti-inflammatory pain, painkillers and pau d’arco.